A MAPK-positive feedback mechanism for BLR1 signaling propels retinoic acid-triggered differentiation and cell cycle arrest (Q38296404)

1 September 2017

A MAPK-positive feedback mechanism for BLR1 signaling propels retinoic acid-triggered differentiation and cell cycle arrest (English)

1 reference

1 September 2017

Jianrong Wang

1

1 reference

PubMed ID

18006504

1 September 2017

Andrew Yen

series ordinal

2

1 reference

PubMed ID

18006504

1 September 2017

language of work or name

English

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publication date

15 November 2007

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Journal of Biological Chemistry

1 September 2017

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1 September 2017

283

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1 September 2017

7

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1 September 2017

4375-4386

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Retinoic acid increases amount of phosphorylated RAF; ectopic expression of cFMS reveals that retinoic acid-induced differentiation is more strongly dependent on ERK2 signaling than induced GO arrest is.

1 September 2017

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Differentiation-specific expression of a novel G protein-coupled receptor from Burkitt's lymphoma

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

The G protein-coupled receptor BLR1 is involved in murine B cell differentiation and is also expressed in neuronal tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

A putative chemokine receptor, BLR1, directs B cell migration to defined lymphoid organs and specific anatomic compartments of the spleen

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Retinoic acid-induced blr1 expression requires RARalpha, RXR, and MAPK activation and uses ERK2 but not JNK/SAPK to accelerate cell differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

A novel retinoic acid-responsive element regulates retinoic acid-induced BLR1 expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Retinoic acid causes MEK-dependent RAF phosphorylation through RARalpha plus RXR activation in HL-60 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Sustained activation of the mitogen-activated protein (MAP) kinase cascade may be required for differentiation of PC12 cells. Comparison of the effects of nerve growth factor and epidermal growth factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Retinoic acid-induced CD38 expression in HL-60 myeloblastic leukemia cells regulates cell differentiation or viability depending on expression levels.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

All-trans retinoic acid induces p62DOK1 and p56DOK2 expression which enhances induced differentiation and G0 arrest of HL-60 leukemia cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Dependence of HL-60 myeloid cell differentiation on continuous and split retinoic acid exposures: precommitment memory associated with altered nuclear structure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Integrin-mediated adhesion regulates ERK nuclear translocation and phosphorylation of Elk-1

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Nuclear localization and regulation of erk- and rsk-encoded protein kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Phosphorylation of transcription factor p62TCF by MAP kinase stimulates ternary complex formation at c-fos promoter

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Growth factors induce nuclear translocation of MAP kinases (p42mapk and p44mapk) but not of their activator MAP kinase kinase (p45mapkk) in fibroblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Meaningful relationships: the regulation of the Ras/Raf/MEK/ERK pathway by protein interactions

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

p21WAF1/CIP1 is a common transcriptional target of retinoid receptors: pleiotropic regulatory mechanism through retinoic acid receptor (RAR)/retinoid X receptor (RXR) heterodimer and RXR/RXR homodimer

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Raf-1 promotes cell survival by antagonizing apoptosis signal-regulating kinase 1 through a MEK-ERK independent mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Autoregulation of the Raf-1 serine/threonine kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

The ins and outs of Raf kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Mutational activation of c-raf-1 and definition of the minimal transforming sequence

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Definition of the human raf amino-terminal regulatory region by deletion mutagenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Negative regulation of Raf-1 by phosphorylation of serine 621.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Regulation of Raf-1 by direct feedback phosphorylation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

14-3-3 is not essential for Raf-1 function: identification of Raf-1 proteins that are biologically activated in a 14-3-3- and Ras-independent manner

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

14-3-3 proteins are required for maintenance of Raf-1 phosphorylation and kinase activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

Ras-independent Activation of the Raf/MEK/ERK Pathway upon Calcium-induced Differentiation of Keratinocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M708471200

21 January 2018

Identifiers

DOI

10.1074/JBC.M708471200

1 reference

1 September 2017

1 reference

PubMed ID

18006504