Integrative analysis reveals 53BP1 copy loss and decreased expression in a subset of human diffuse large B-cell lymphomas (Q38299935)

1 September 2017

Integrative analysis reveals 53BP1 copy loss and decreased expression in a subset of human diffuse large B-cell lymphomas (English)

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1 September 2017

5

G Getz

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6

R Beroukhim

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K Takeyama

1

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S Monti

2

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J P Manis

3

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P Dal Cin

4

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S Dutt

7

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J C Aster

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F W Alt

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T R Golub

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M A Shipp

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language of work or name

English

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publication date

16 July 2007

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27

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3

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318-322

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1210650

1 September 2017

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Integrative genomic analyses identify MITF as a lineage survival oncogene amplified in malignant melanoma

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Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

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53BP1 links DNA damage-response pathways to immunoglobulin heavy chain class-switch recombination.

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Common deletion polymorphisms in the human genome.

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53BP1 and p53 synergize to suppress genomic instability and lymphomagenesis.

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The BCL6 proto-oncogene suppresses p53 expression in germinal-centre B cells

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BCL6 interacts with the transcription factor Miz-1 to suppress the cyclin-dependent kinase inhibitor p21 and cell cycle arrest in germinal center B cells

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Transcriptional signature with differential expression of BCL6 target genes accurately identifies BCL6-dependent diffuse large B cell lymphomas.

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Global variation in copy number in the human genome

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A direct approach to false discovery rates

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53BP1 cooperates with p53 and functions as a haploinsufficient tumor suppressor in mice

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Accumulation of Checkpoint Protein 53BP1 at DNA Breaks Involves Its Binding to Phosphorylated Histone H2AX

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p53 Binding protein 53BP1 is required for DNA damage responses and tumor suppression in mice

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Molecular profiling of diffuse large B-cell lymphoma identifies robust subtypes including one characterized by host inflammatory response

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FAS Death Domain Deletions and Cellular FADD-like Interleukin 1 Converting Enzyme Inhibitory Protein (Long) Overexpression: Alternative Mechanisms for Deregulating the Extrinsic Apoptotic Pathway in Diffuse Large B-Cell Lymphoma Subtypes

21 January 2018

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A high-resolution survey of deletion polymorphism in the human genome

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inferred from DOI database lookup

7 January 2021

based on heuristic

53BP1, an activator of ATM in response to DNA damage

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1210650

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.ONC.1210650

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Dimensions Publication ID

1 September 2017

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