SADS: A new component of Fas-DISC is the accelerator for cell death signaling and is downregulated in patients with colon carcinoma (Q38305316)

Dataset for "Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine"

2 September 2017

3 references

Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

PubMed

https://github.com/jmillanacosta/retractable/blob/e28dcac33244e89948fe17fb65f09b59e2ed804d/data/PMIDS_DOIS.csv

title

SADS: A new component of Fas-DISC is the accelerator for cell death signaling and is downregulated in patients with colon carcinoma (English)

1 reference

2 September 2017

1 reference

Suzuki A

1

1 reference

2 September 2017

Obata S

2

1 reference

2 September 2017

Hayashida M

3

1 reference

2 September 2017

Kawano H

4

1 reference

2 September 2017

Nakano T

5

1 reference

2 September 2017

Shiraki K

6

1 reference

2 September 2017

language of work or name

English

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publication date

1 January 2001

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2 September 2017

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2 September 2017

volume

7

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2 September 2017

issue

1

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2 September 2017

page(s)

88-93

1 reference

Cell death: the significance of apoptosis

2 September 2017

cites work

1 reference

7 January 2021

Apoptosis by death factor

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7 January 2021

1 reference

7 January 2021

A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor

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7 January 2021

Human ICE/CED-3 protease nomenclature

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7 January 2021

Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosis

1 reference

7 January 2021

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

1 reference

7 January 2021

The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

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7 January 2021

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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7 January 2021

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

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7 January 2021

A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis

1 reference

7 January 2021

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

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7 January 2021

Acinus is a caspase-3-activated protein required for apoptotic chromatin condensation

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7 January 2021

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

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7 January 2021

Expression of Fas ligand in liver metastases of human colonic adenocarcinomas

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7 January 2021

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

7 January 2021

The CED-4-homologous protein FLASH is involved in Fas-mediated activation of caspase-8 during apoptosis

1 reference

7 January 2021

FADD/MORT1 and caspase-8 are recruited to TRAIL receptors 1 and 2 and are essential for apoptosis mediated by TRAIL receptor 2

1 reference

7 January 2021

Apo2L/TRAIL-dependent recruitment of endogenous FADD and caspase-8 to death receptors 4 and 5.

1 reference

7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

1 reference

7 January 2021

Studies of the lamin proteinase reveal multiple parallel biochemical pathways during apoptotic execution

1 reference

30 December 2023

https://opencitations.net/index/api/v1/references/10.1038/83401

Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor

1 reference

30 December 2023

https://opencitations.net/index/api/v1/references/10.1038/83401

Functional absence of FADD in PLC/PRF/5 hepatoma cells: possible involvement in the transformation to hepatoma in HBV-infected hepatocytes.

1 reference

30 December 2023

https://opencitations.net/index/api/v1/references/10.1038/83401

Identifiers

DOI

10.1038/83401

1 reference

2 September 2017

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