Nonsteroidal anti-inflammatory drugs suppress cancer stem cells via inhibiting PTGS2 (cyclooxygenase 2) and NOTCH/HES1 and activating PPARG in colorectal cancer. (Q38448721)

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scientific article published on 07 August 2013

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English	Nonsteroidal anti-inflammatory drugs suppress cancer stem cells via inhibiting PTGS2 (cyclooxygenase 2) and NOTCH/HES1 and activating PPARG in colorectal cancer.	scientific article published on 07 August 2013

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scholarly article

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Nonsteroidal anti-inflammatory drugs suppress cancer stem cells via inhibiting PTGS2 (cyclooxygenase 2) and NOTCH/HES1 and activating PPARG in colorectal cancer (English)

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colorectal cancer

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based on heuristic

inferred from title

neoplastic stem cells

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based on heuristic

inferred from title

8

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Chang Mo Moon

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Ji-Hee Kwon

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Ji Suk Kim

3

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Sun-Hee Oh

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Kyoung Jin Lee

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Jae Jun Park

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Sung Pil Hong

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Tae Il Kim

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Won Ho Kim

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7 August 2013

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International Journal of Cancer

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134

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3

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519-529

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5 March 2020

Estimates of worldwide burden of cancer in 2008: GLOBOCAN 2008

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Tumour-initiating cells: challenges and opportunities for anticancer drug discovery

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Stem cells, cancer, and cancer stem cells

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Human colon cancer stem cells: a new paradigm in gastrointestinal oncology

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Phenotypic characterization of human colorectal cancer stem cells

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CD133+CD44+ population efficiently enriches colon cancer initiating cells

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Colorectal cancer stem cells are enriched in xenogeneic tumors following chemotherapy

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Chemoresistant colorectal cancer cells, the cancer stem cell phenotype, and increased sensitivity to insulin-like growth factor-I receptor inhibition

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Symmetric division of cancer stem cells--a key mechanism in tumor growth that should be targeted in future therapeutic approaches

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Truncated mammalian Notch1 activates CBF1/RBPJk-repressed genes by a mechanism resembling that of Epstein-Barr virus EBNA2

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15-Deoxy-delta 12, 14-prostaglandin J2 is a ligand for the adipocyte determination factor PPAR gamma

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Regulation of Notch1/NICD and Hes1 expressions by GSK-3alpha/beta

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The COX-2 selective inhibitor-independent COX-2 effect on colon carcinoma cells is associated with the Delta1/Notch1 pathway

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Selected non-steroidal anti-inflammatory drugs and their derivatives target gamma-secretase at a novel site. Evidence for an allosteric mechanism.

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Peroxisome proliferator-activated receptors alpha and gamma are activated by indomethacin and other non-steroidal anti-inflammatory drugs

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The role of NF-κB and PPARγ in experimentally induced colorectal cancer and chemoprevention by cyclooxygenase-2 inhibitors.

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A synthetic antagonist for the peroxisome proliferator-activated receptor gamma inhibits adipocyte differentiation

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A randomized trial of aspirin to prevent colorectal adenomas

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Long-term use of aspirin and nonsteroidal anti-inflammatory drugs and risk of colorectal cancer

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Sulindac suppresses tumorigenesis in the Min mouse.

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Up-regulation of cyclooxygenase 2 gene expression in human colorectal adenomas and adenocarcinomas

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Prostaglandin E2 promotes intestinal tumor growth via DNA methylation

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The role of COX-2 in intestinal inflammation and colorectal cancer

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Suppression of intestinal polyposis in Apc delta716 knockout mice by inhibition of cyclooxygenase 2 (COX-2)

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Prostaglandin E2 promotes colon cancer cell growth through a Gs-axin-beta-catenin signaling axis.

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Targeting Wnt signaling: can we safely eradicate cancer stem cells?

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Role of COX-independent targets of NSAIDs and related compounds in cancer prevention and treatment

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Inhibition of NF-kappaB and AP-1 activation by R- and S-flurbiprofen

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Chemoprevention by nonsteroidal anti-inflammatory drugs eliminates oncogenic intestinal stem cells via SMAC-dependent apoptosis

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21 January 2018

NOTCH signaling is required for formation and self-renewal of tumor-initiating cells and for repression of secretory cell differentiation in colon cancer

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PPARgamma agonists inhibit growth and expansion of CD133+ brain tumour stem cells

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Cyclooxygenase-2 inhibitors prevent trinitrobenzene sulfonic acid-induced P-glycoprotein up-regulation in vitro and in vivo

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Identifiers

10.1002/IJC.28381

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