TIS21/BTG2/PC3 is expressed through PKC-delta pathway and inhibits binding of cyclin B1-Cdc2 and its activity, independent of p53 expression. (Q38516758)

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scientific article published on September 2004

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English	TIS21/BTG2/PC3 is expressed through PKC-delta pathway and inhibits binding of cyclin B1-Cdc2 and its activity, independent of p53 expression.	scientific article published on September 2004

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31 December 2019

TIS21/BTG2/PC3 is expressed through PKC-delta pathway and inhibits binding of cyclin B1-Cdc2 and its activity, independent of p53 expression (English)

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31 December 2019

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31 December 2019

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Min Sook Ryu

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31 December 2019

Myung Soog Lee

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31 December 2019

Jong Wook Hong

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31 December 2019

Tae-Ryong Hahn

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31 December 2019

Eunpyo Moon

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1 September 2004

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31 December 2019

Experimental Cell Research

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31 December 2019

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31 December 2019

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31 December 2019

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31 December 2019

Primary response genes induced by growth factors and tumor promoters

1 reference

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p53, the cellular gatekeeper for growth and division

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Mitogen-stimulated TIS21 protein interacts with a protein-kinase-Calpha-binding protein rPICK1

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Immunocytochemical localization of eight protein kinase C isozymes overexpressed in NIH 3T3 fibroblasts. Isoform-specific association with microfilaments, Golgi, endoplasmic reticulum, and nuclear and cell membranes

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Antiproliferative proteins of the BTG/Tob family are degraded by the ubiquitin-proteasome system

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Synthesis, degradation, and subcellular localization of proteins encoded by the primary response genes TIS7/PC4 and TIS21/PC3

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Differential effects of phorbol ester on signaling and gene expression in human leukemia cells

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Activation of protein kinase C relays distinct signaling pathways in the same cell type: differentiation and caspase-mediated apoptosis

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Cyclin A is destroyed in prometaphase and can delay chromosome alignment and anaphase

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Animal cell cycles and their control

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BTG gene expression in the p53-dependent and -independent cellular response to DNA damage

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Alternative reading frames of the INK4a tumor suppressor gene encode two unrelated proteins capable of inducing cell cycle arrest

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