High mechanical strain of primary intervertebral disc cells promotes secretion of inflammatory factors associated with disc degeneration and pain. (Q38652746)

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English	High mechanical strain of primary intervertebral disc cells promotes secretion of inflammatory factors associated with disc degeneration and pain.	scientific article

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6 March 2020

High mechanical strain of primary intervertebral disc cells promotes secretion of inflammatory factors associated with disc degeneration and pain (English)

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intervertebral disc cell

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Derek H Rosenzweig

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Jean A Ouellet

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Laura S Stone

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Thomas M Quinn

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Lisbet Haglund

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23 January 2014

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Arthritis Research and Therapy

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R21

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1

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CZ Software Mentions: A large dataset of software mentions in the biomedical literature

https://scigraph.springernature.com/pub.10.1186/ar4449

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Inflammatory mediators in intervertebral disk degeneration and discogenic pain

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Culture of primary bovine chondrocytes on a continuously expanding surface inhibits dedifferentiation

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Spinal TLR4 mediates the transition to a persistent mechanical hypersensitivity after the resolution of inflammation in serum-transferred arthritis

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The rat intervertebral disk degeneration pain model: relationships between biological and structural alterations and pain

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TNF-α and IL-1β promote a disintegrin-like and metalloprotease with thrombospondin type I motif-5-mediated aggrecan degradation through syndecan-4 in intervertebral disc

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Mild-stretch mechanical ventilation upregulates toll-like receptor 2 and sensitizes the lung to bacterial lipopeptide

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Response of chondrocytes to shear stress: antagonistic effects of the binding partners Toll-like receptor 4 and caveolin-1.

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6 September 2017

Cyclic stretch-induced apoptosis in rat annulus fibrosus cells is mediated in part by endoplasmic reticulum stress through nitric oxide production

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The involvement of interleukin-1 and interleukin-4 in the response of human annulus fibrosus cells to cyclic tensile strain: an altered mechanotransduction pathway with degeneration

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Validation of the cat as a model for the human lumbar spine during simulated high-velocity, low-amplitude spinal manipulation

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Chondrocyte innate immune myeloid differentiation factor 88-dependent signaling drives procatabolic effects of the endogenous Toll-like receptor 2/Toll-like receptor 4 ligands low molecular weight hyaluronan and high mobility group box chromosomal p

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Proteomic analysis of the LPS-induced stress response in rat chondrocytes reveals induction of innate immune response components in articular cartilage

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Catabolic cytokine expression in degenerate and herniated human intervertebral discs: IL-1beta and TNFalpha expression profile.

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What is intervertebral disc degeneration, and what causes it?

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Innervation, inflammation, and hypermobility may characterize pathologic disc degeneration: review of animal model data

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High strain mechanical loading rapidly induces tendon apoptosis: an ex vivo rat tibialis anterior model.

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Toll-like receptor signalling

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TLR signaling pathways

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A role for TNFα in intervertebral disc degeneration: a non-recoverable catabolic shift.

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Sustained loading generates stress concentrations in lumbar intervertebral discs.

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Surface strain on human intervertebral discs.

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[In vivo study of innervation of degenerative intervertebral discs in rabbit anular-injury model]

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1 reference

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6 March 2020

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