The newly synthesized 2-arylnaphthyridin-4-one, CSC-3436, induces apoptosis of non-small cell lung cancer cells by inhibiting tubulin dynamics and activating CDK1. (Q38878480)

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scientific article published on 7 May 2015

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English	The newly synthesized 2-arylnaphthyridin-4-one, CSC-3436, induces apoptosis of non-small cell lung cancer cells by inhibiting tubulin dynamics and activating CDK1.	scientific article published on 7 May 2015

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7 March 2020

The newly synthesized 2-arylnaphthyridin-4-one, CSC-3436, induces apoptosis of non-small cell lung cancer cells by inhibiting tubulin dynamics and activating CDK1 (English)

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7 March 2020

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7 March 2020

Ruey-Hwang Chou

5

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7 March 2020

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7 March 2020

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Ling-Chu Chang

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7 March 2020

Yung-Luen Yu

2

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7 March 2020

Chin-Yu Liu

3

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7 March 2020

Min-Tsang Hsieh

6

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7 March 2020

Hui-Yi Lin

7

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7 March 2020

Hsin-Yi Hung

8

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7 March 2020

Li-Jiau Huang

9

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7 March 2020

Sheng-Chu Kuo

11

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7 March 2020

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7 May 2015

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7 March 2020

Cancer Chemotherapy and Pharmacology

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7 March 2020

75

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7 March 2020

6

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7 March 2020

1303-1315

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7 March 2020

Cancer statistics, 2015

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On and around microtubules: an overview

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Differential Apaf-1 levels allow cytochrome c to induce apoptosis in brain tumors but not in normal neural tissues

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Microtubule-targeted anticancer agents and apoptosis

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Microtubules in apoptosis induction: are they necessary?

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Phosphorylation of caspase-9 by CDK1/cyclin B1 protects mitotic cells against apoptosis

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CAK-Cyclin-dependent Activating Kinase: a key kinase in cell cycle control and a target for drugs?

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Mammalian cyclin-dependent kinases

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Cyclin-dependent kinase-1: linking apoptosis to cell cycle and mitotic catastrophe.

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Cyclin-dependent kinase-1 (Cdk1)/cyclin B1 dictates cell fate after mitotic arrest via phosphoregulation of antiapoptotic Bcl-2 proteins

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Two mitosis-specific antibodies, MPM-2 and phospho-histone H3 (Ser28), allow rapid and precise determination of mitotic activity.

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DNA breaks and chromosome pulverization from errors in mitosis

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Taxol-induced apoptosis depends on MAP kinase pathways (ERK and p38) and is independent of p53.

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Role of JNK activation in apoptosis: a double-edged sword

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Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

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JNK-mediated phosphorylation of Cdc25C regulates cell cycle entry and G(2)/M DNA damage checkpoint.

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Cdk1/cyclin B plays a key role in mitotic arrest-induced apoptosis by phosphorylation of Mcl-1, promoting its degradation and freeing Bak from sequestration

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10.1007/S00280-015-2765-0

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7 March 2020

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7 March 2020

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