miR-30c Mediates Upregulation of Cdc42 and Pak1 in Diabetic Cardiomyopathy. (Q38898539)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25781190%20AND%20SRC:MED&resulttype=core&format=json

3 March 2020

title

miR-30c Mediates Upregulation of Cdc42 and Pak1 in Diabetic Cardiomyopathy (English)

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3 March 2020

8

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3 March 2020

author name string

Satish K Raut

1

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3 March 2020

Akhilesh Kumar

2

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3 March 2020

Gurinder B Singh

3

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3 March 2020

Uma Nahar

4

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3 March 2020

Vibhuti Sharma

5

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3 March 2020

Anupam Mittal

6

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3 March 2020

Rajni Sharma

7

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3 March 2020

publication date

1 June 2015

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Cardiovascular Therapeutics

3 March 2020

published in

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3 March 2020

volume

33

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3 March 2020

issue

3

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3 March 2020

page(s)

89-97

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Myosin heads are displaced from actin filaments in the in situ beating rat heart in early diabetes

3 March 2020

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The mechanism of PAK activation. Autophosphorylation events in both regulatory and kinase domains control activity

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MicroRNA-133 controls cardiac hypertrophy

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Regulation of mitogen-activated protein kinases in cardiac myocytes through the small G protein Rac1

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Enhanced activation of p21-activated kinase 1 in heart failure contributes to dephosphorylation of connexin 43.

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Cdc42 is an antihypertrophic molecular switch in the mouse heart

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The prevention of diabetic cardiomyopathy by non-mitogenic acidic fibroblast growth factor is probably mediated by the suppression of oxidative stress and damage

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miR133a regulates cardiomyocyte hypertrophy in diabetes.

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MicroRNA deregulation in right ventricular outflow tract myocardium in nonsyndromic tetralogy of fallot

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TGFβR2 is a major target of miR-93 in nasopharyngeal carcinoma aggressiveness

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Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy

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Cdc42 plays a critical role in assembly of sarcomere units in series of cardiac myocytes

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A brain serine/threonine protein kinase activated by Cdc42 and Rac1

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Ablation of p21-activated kinase-1 in mice promotes isoproterenol-induced cardiac hypertrophy in association with activation of Erk1/2 and inhibition of protein phosphatase 2A

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Small guanine nucleotide-binding proteins and myocardial hypertrophy

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Heregulin regulates cytoskeletal reorganization and cell migration through the p21-activated kinase-1 via phosphatidylinositol-3 kinase

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MicroRNAs are aberrantly expressed in hypertrophic heart: do they play a role in cardiac hypertrophy?

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Regulation of Akt/PKB activity by P21-activated kinase in cardiomyocytes

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Cytoskeletal role in protection of the failing heart by β-adrenergic blockade

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Pak1 as a novel therapeutic target for antihypertrophic treatment in the heart

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miR-133 and miR-30 regulate connective tissue growth factor: implications for a role of microRNAs in myocardial matrix remodeling

21 January 2018

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MiR-30-regulated autophagy mediates angiotensin II-induced myocardial hypertrophy

21 January 2018

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A phenotypic screen to identify hypertrophy-modulating microRNAs in primary cardiomyocytes

21 January 2018

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