Epithelial-to-mesenchymal transition (EMT) induced by inflammatory priming elicits mesenchymal stromal cell-like immune-modulatory properties in cancer cells. (Q38911110)

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English	Epithelial-to-mesenchymal transition (EMT) induced by inflammatory priming elicits mesenchymal stromal cell-like immune-modulatory properties in cancer cells.	scientific article

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29 February 2020

Epithelial-to-mesenchymal transition (EMT) induced by inflammatory priming elicits mesenchymal stromal cell-like immune-modulatory properties in cancer cells (English)

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29 February 2020

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Mario Ricciardi

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29 February 2020

Giorgio Malpeli

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29 February 2020

Omar Perbellini

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29 February 2020

Francesco Bifari

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29 February 2020

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M Zanotto

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29 February 2020

G Bassi

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29 February 2020

M Chilosi

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29 February 2020

M Krampera

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29 February 2020

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17 March 2015

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29 February 2020

British Journal of Cancer

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29 February 2020

112

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29 February 2020

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29 February 2020

1067-1075

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29 February 2020

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Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International

10 February 2015

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April 2022 Public Data File from Crossref

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0 references

https://scigraph.springernature.com/pub.10.1038/bjc.2015.29

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Mesenchymal stem cells use IDO to regulate immunity in tumor microenvironment

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9 September 2017

Tryptophan catabolism in cancer: beyond IDO and tryptophan depletion

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9 September 2017

Autophagy and apoptosis-related genes in chronic liver disease and hepatocellular carcinoma.

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9 September 2017

Mesenchymal stromal cell 'licensing': a multistep process

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9 September 2017

Tumor-evoked regulatory B cells promote breast cancer metastasis by converting resting CD4⁺ T cells to T-regulatory cells

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9 September 2017

miR-661 expression in SNAI1-induced epithelial to mesenchymal transition contributes to breast cancer cell invasion by targeting Nectin-1 and StarD10 messengers

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9 September 2017

Immunity, Inflammation, and Cancer

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9 September 2017

Inflammation and EMT: an alliance towards organ fibrosis and cancer progression

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9 September 2017

Epithelial-mesenchymal transitions in development and disease

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9 September 2017

Inhibitors of indoleamine-2,3-dioxygenase for cancer therapy: can we see the wood for the trees?

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9 September 2017

Epithelial-mesenchymal transition: a cancer researcher's conceptual friend and foe.

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9 September 2017

Epithelial-mesenchymal transition in cancer metastasis: mechanisms, markers and strategies to overcome drug resistance in the clinic

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9 September 2017

Cancer-related inflammation

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9 September 2017

Classical and nonclassical HLA class I antigen and NK Cell-activating ligand changes in malignant cells: current challenges and future directions

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9 September 2017

Inhibition of indoleamine 2,3-dioxygenase, an immunoregulatory target of the cancer suppression gene Bin1, potentiates cancer chemotherapy.

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4366889

9 September 2017

Human T regulatory cells can use the perforin pathway to cause autologous target cell death

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9 September 2017

Evidence for a tumoral immune resistance mechanism based on tryptophan degradation by indoleamine 2,3-dioxygenase

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Mechanism for elimination of a tumor suppressor: aberrant splicing of a brain-specific exon causes loss of function of Bin1 in melanoma.

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Antiestrogens induce transforming growth factor beta-mediated immunosuppression in breast cancer

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14 September 2017

Cancer metastasis is accelerated through immunosuppression during Snail-induced EMT of cancer cells

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14 September 2017

IDO1 and IDO2 are expressed in human tumors: levo- but not dextro-1-methyl tryptophan inhibits tryptophan catabolism

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Expression of indoleamine 2,3-dioxygenase in tumor endothelial cells correlates with long-term survival of patients with renal cell carcinoma

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Foxp3 expressing CD4+CD25(high) regulatory T cells are overrepresented in human metastatic melanoma lymph nodes and inhibit the function of infiltrating T cells

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Expression of a MYCN-interacting isoform of the tumor suppressor BIN1 is reduced in neuroblastomas with unfavorable biological features.

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22 August 2018

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A cell culture model for T lymphocyte clonal anergy

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12 December 2020

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Losses of the tumor suppressor BIN1 in breast carcinoma are frequent and reflect deficits in programmed cell death capacity

1 reference

https://pubmed.ncbi.nlm.nih.gov/25668006

12 December 2020

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Loss of heterozygosity and tumor suppressor activity of Bin1 in prostate carcinoma

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https://pubmed.ncbi.nlm.nih.gov/25668006

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based on heuristic

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10.1038/BJC.2015.29

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29 February 2020

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29 February 2020

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29 February 2020

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