MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1. (Q38987204)

8 September 2017

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1. (English)

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8 September 2017

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Feng He

1

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8 September 2017

Fenfen Peng

2

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8 September 2017

Xi Xia

3

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8 September 2017

Chen Zhao

4

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8 September 2017

Qimei Luo

5

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8 September 2017

Weiming Guan

6

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8 September 2017

Zhijian Li

7

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8 September 2017

Xueqing Yu

8

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8 September 2017

Fengxian Huang

9

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8 September 2017

9 June 2014

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8 September 2017

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8 September 2017

57

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8 September 2017

1726-1736

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8 September 2017

8

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https://scigraph.springernature.com/pub.10.1007/s00125-014-3282-0

8 September 2017

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HNF4 alpha and the Ca-channel TRPC1 are novel disease candidate genes in diabetic nephropathy

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Evaluation of genetic association and expression reduction of TRPC1 in the development of diabetic nephropathy

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Association of TRPC1 gene polymorphisms with type 2 diabetes and diabetic nephropathy in Han Chinese population

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STIM1 carboxyl-terminus activates native SOC, I(crac) and TRPC1 channels

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3-Hydroxy-3-methylglutaryl CoA reductase inhibitors prevent high glucose-induced proliferation of mesangial cells via modulation of Rho GTPase/ p21 signaling pathway: Implications for diabetic nephropathy

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Protein kinase Calpha phosphorylates the TRPC1 channel and regulates store-operated Ca2+ entry in endothelial cells

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Diabetic retinopathy in predicting diabetic nephropathy in patients with type 2 diabetes and renal disease: a meta-analysis

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PubMed

https://pubmed.ncbi.nlm.nih.gov/24908566

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1007/S00125-014-3282-0

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Dimensions Publication ID

8 September 2017

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