Serum amyloid A induces interleukin-6 in dermal fibroblasts via Toll-like receptor 2, interleukin-1 receptor-associated kinase 4 and nuclear factor-κB. (Q39029262)

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scientific article published on November 2014

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English	Serum amyloid A induces interleukin-6 in dermal fibroblasts via Toll-like receptor 2, interleukin-1 receptor-associated kinase 4 and nuclear factor-κB.	scientific article published on November 2014

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scholarly article

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6 March 2020

Serum amyloid A induces interleukin-6 in dermal fibroblasts via Toll-like receptor 2, interleukin-1 receptor-associated kinase 4 and nuclear factor-κB (English)

1 reference

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6 March 2020

toll-like receptor

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Marzena Ciechomska

3

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6 March 2020

4

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6 March 2020

author name string

Steven O'Reilly

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6 March 2020

Rachel Cant

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6 March 2020

Fiona Oakley

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6 March 2020

Sophie Hambleton

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6 March 2020

Jacob M van Laar

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6 March 2020

publication date

1 November 2014

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6 March 2020

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6 March 2020

143

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6 March 2020

3

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6 March 2020

331-340

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6 March 2020

Role of toll-like receptors in systemic sclerosis.

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Acute-phase serum amyloid a in osteoarthritis: regulatory mechanism and proinflammatory properties

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Upregulation of Toll-like receptor 2 gene expression by acetylation of AP-2 alpha in THP-1 cells, a human monocytic cell line

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Toll-like receptor-mediated, enhanced production of profibrotic TIMP-1 in monocytes from patients with systemic sclerosis: role of serum factors.

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Tumor necrosis factor-costimulated T lymphocytes from patients with systemic sclerosis trigger collagen production in fibroblasts.

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15 October 2018

Serum amyloid A triggers the mosodium urate -mediated mature interleukin-1β production from human synovial fibroblasts

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T cells in systemic sclerosis: a reappraisal

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Association of C-reactive protein with high disease activity in systemic sclerosis: results from the Canadian Scleroderma Research Group

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Inhibition of signal transducer and activator of transcription 3 (STAT3) attenuates interleukin-6 (IL-6)-induced collagen synthesis and resultant hypertrophy in rat heart.

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Acute-phase serum amyloid A regulates tumor necrosis factor α and matrix turnover and predicts disease progression in patients with inflammatory arthritis before and after biologic therapy.

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High mobility group box protein 1 in complex with lipopolysaccharide or IL-1 promotes an increased inflammatory phenotype in synovial fibroblasts.

1 reference

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Serum amyloid A activates the NLRP3 inflammasome via P2X7 receptor and a cathepsin B-sensitive pathway

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15 October 2018

Serum amyloid A overrides Treg anergy via monocyte-dependent and Treg-intrinsic, SOCS3-associated pathways

1 reference

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Serum amyloid A induces reactive oxygen species (ROS) production and proliferation of fibroblast

1 reference

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15 October 2018

Toll-like receptors expressed by dermal fibroblasts contribute to hypertrophic scarring.

1 reference

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Fibrosis and immune dysregulation in systemic sclerosis

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15 October 2018

Could antibodies against serum amyloid A function as physiological regulators in humans?

1 reference

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Circulating mitochondrial DAMPs cause inflammatory responses to injury

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Bronchial epithelial cells cultured from clinically stable lung allograft patients promote the development of macrophages from monocytes rather than dendritic cells.

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Serum amyloid A induces G-CSF expression and neutrophilia via Toll-like receptor 2

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Cutting edge: TLR2 is a functional receptor for acute-phase serum amyloid A

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Serum amyloid A binding to formyl peptide receptor-like 1 induces synovial hyperplasia and angiogenesis.

1 reference

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How Toll-like receptors signal: what we know and what we don't know

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Matrix contraction by dermal fibroblasts requires transforming growth factor-beta/activin-linked kinase 5, heparan sulfate-containing proteoglycans, and MEK/ERK: insights into pathological scarring in chronic fibrotic disease

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Involvement of toll-like receptors 2 and 4 in cellular activation by high mobility group box 1 protein

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Lipopolysaccharide binding protein and serum amyloid A secretion by human intestinal epithelial cells during the acute phase response

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Regulation of serum amyloid A protein expression during the acute-phase response

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Serum amyloid A protein concentration in progressive systemic sclerosis (scleroderma)

1 reference

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Hypoxia-induced increase in the production of extracellular matrix proteins in systemic sclerosis

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1 reference

https://pubmed.ncbi.nlm.nih.gov/24476318

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Enhanced production of interleukin-6 (IL-6), oncostatin M and soluble IL-6 receptor by cultured peripheral blood mononuclear cells from patients with systemic sclerosis

1 reference

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12 December 2020

based on heuristic

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