Reactive oxygen species are induced by Kaposi's sarcoma-associated herpesvirus early during primary infection of endothelial cells to promote virus entry. (Q39240333)

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scientific article published on 21 November 2012

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English	Reactive oxygen species are induced by Kaposi's sarcoma-associated herpesvirus early during primary infection of endothelial cells to promote virus entry.	scientific article published on 21 November 2012

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18 February 2020

Reactive oxygen species are induced by Kaposi's sarcoma-associated herpesvirus early during primary infection of endothelial cells to promote virus entry (English)

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18 February 2020

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reactive oxygen species

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based on heuristic

inferred from title

Sayan Chakraborty

2

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18 February 2020

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Virginie Bottero

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18 February 2020

Bala Chandran

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21 November 2012

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18 February 2020

Journal of Virology

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18 February 2020

87

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18 February 2020

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18 February 2020

1733-1749

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18 February 2020

A role for virally induced reactive oxygen species in Kaposi's sarcoma herpesvirus tumorigenesis

1 reference

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10 September 2017

The ephrin receptor tyrosine kinase A2 is a cellular receptor for Kaposi's sarcoma–associated herpesvirus

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10 September 2017

Kaposi's sarcoma-associated herpesvirus interacts with EphrinA2 receptor to amplify signaling essential for productive infection

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Reactive oxygen species drive herpes simplex virus (HSV)-1-induced proinflammatory cytokine production by murine microglia

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c-Cbl-mediated selective virus-receptor translocations into lipid rafts regulate productive Kaposi's sarcoma-associated herpesvirus infection in endothelial cells.

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Adenovirus type 5 rupture of lysosomes leads to cathepsin B-dependent mitochondrial stress and production of reactive oxygen species.

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Reactive oxygen species hydrogen peroxide mediates Kaposi's sarcoma-associated herpesvirus reactivation from latency

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Interaction of c-Cbl with myosin IIA regulates Bleb associated macropinocytosis of Kaposi's sarcoma-associated herpesvirus

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Kaposi's sarcoma associated herpes virus (KSHV) induced COX-2: a key factor in latency, inflammation, angiogenesis, cell survival and invasion

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Early events in Kaposi's sarcoma-associated herpesvirus infection of target cells

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Antitumorigenesis of antioxidants in a transgenic Rac1 model of Kaposi's sarcoma

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The Epstein-Barr virus nuclear antigen-1 promotes genomic instability via induction of reactive oxygen species.

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10 September 2017

Kaposi's Sarcoma-Associated Herpesvirus Forms a Multimolecular Complex of Integrins ( V 5, V 3, and 3 1) and CD98-xCT during Infection of Human Dermal Microvascular Endothelial Cells, and CD98-xCT Is Essential for the Postentry Stage of Infection

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The role of reactive oxygen species in integrin and matrix metalloproteinase expression and function

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The antiapoptotic herpes simplex virus glycoprotein J localizes to multiple cellular organelles and induces reactive oxygen species formation

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Kaposi's sarcoma-associated herpesvirus fusion-entry receptor: cystine transporter xCT.

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Kaposi's sarcoma-associated herpesvirus induction of AP-1 and interleukin 6 during primary infection mediated by multiple mitogen-activated protein kinase pathways

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Reactive oxygen signaling and MAPK activation distinguish Epstein-Barr Virus (EBV)-positive versus EBV-negative Burkitt's lymphoma

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Integrins and coagulation: a role for ROS/redox signaling?

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Concurrent expression of latent and a limited number of lytic genes with immune modulation and antiapoptotic function by Kaposi's sarcoma-associated herpesvirus early during infection of primary endothelial and fibroblast cells and subsequent declin

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Tiam1 mediates neurite outgrowth induced by ephrin-B1 and EphA2

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Kaposi's sarcoma-associated herpesvirus induces the phosphatidylinositol 3-kinase-PKC-zeta-MEK-ERK signaling pathway in target cells early during infection: implications for infectivity

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Regulation of the EphA2 kinase by the low molecular weight tyrosine phosphatase induces transformation

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Integrins engage mitochondrial function for signal transduction by a mechanism dependent on Rho GTPases

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Reactive oxygen species and signal transduction

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Focal adhesion kinase regulation by oxidative stress in different cell types

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Role of Rac1 and oxygen radicals in collagenase-1 expression induced by cell shape change

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Intracellular thiols regulate activation of nuclear factor kappa B and transcription of human immunodeficiency virus

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RhoA-GTPase facilitates entry of Kaposi's sarcoma-associated herpesvirus into adherent target cells in a Src-dependent manner

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Kaposi's sarcoma-associated herpesvirus induces sustained NF-kappaB activation during de novo infection of primary human dermal microvascular endothelial cells that is essential for viral gene expression

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The small GTPase Rac1 links the Kaposi sarcoma-associated herpesvirus vGPCR to cytokine secretion and paracrine neoplasia

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25 September 2017

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15 June 2018

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15 June 2018

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21 August 2018

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21 August 2018

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21 August 2018

Focal adhesion kinase is critical for entry of Kaposi's sarcoma-associated herpesvirus into target cells

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21 August 2018

ERK1/2 and MEK1/2 induced by Kaposi's sarcoma-associated herpesvirus (human herpesvirus 8) early during infection of target cells are essential for expression of viral genes and for establishment of infection

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21 August 2018

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21 August 2018

Reactive oxygen species as essential mediators of cell adhesion: the oxidative inhibition of a FAK tyrosine phosphatase is required for cell adhesion.

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21 August 2018

Oxygen radicals in influenza-induced pathogenesis and treatment with pyran polymer-conjugated SOD

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Reactive oxygen species activate focal adhesion kinase, paxillin and p130cas tyrosine phosphorylation in endothelial cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/23175375

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.02958-12

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Europe PubMed Central

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18 February 2020

1 reference

Europe PubMed Central

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18 February 2020

1 reference

Europe PubMed Central

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18 February 2020

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