Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints (Q39806215)

From Wikidata

Jump to navigation Jump to search

scientific article

Language	Label	Description	Also known as
English	Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints	scientific article

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints (English)

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

0 references

Epstein–Barr virus

0 references

3

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

author name string

B Gruhne

1

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

R Sompallae

2

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

language of work or name

0 references

publication date

31 August 2009

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

28

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

45

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

3997-4008

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Structural and mechanical control of mitotic progression

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

21 January 2018

Effect of the EBNA-2 gene on the surface antigen phenotype of transfected EBV-negative B-lymphoma lines

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

The ATM/p53 pathway is commonly targeted for inactivation in squamous cell carcinoma of the head and neck (SCCHN) by multiple molecular mechanisms

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Down-regulation of ATM protein in HRS cells of nodular sclerosis Hodgkin's lymphoma in children occurs in the absence of ATM gene inactivation.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

The ATM tumour suppressor gene is down-regulated in EBV-associated nasopharyngeal carcinoma.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Reactive oxygen species are downstream products of TRAF-mediated signal transduction

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Mammalian Chk2 is a downstream effector of the ATM-dependent DNA damage checkpoint pathway

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus latent membrane protein 1 represses DNA repair through the PI3K/Akt/FOXO3a pathway in human epithelial cells

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

The ATM/ATR signaling effector Chk2 is targeted by Epstein-Barr virus nuclear antigen 3C to release the G2/M cell cycle block

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Expression of the Epstein-Barr virus (EBV)-encoded membrane antigen (LMP) increases the stimulatory capacity of EBV-negative B lymphoma lines in allogeneic mixed lymphocyte cultures

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Cryptic deletion involving the ATM locus at 11q22.3 approximately q23.1 in B-cell chronic lymphocytic leukemia and related disorders

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

The late lytic LMP-1 protein of Epstein-Barr virus can negatively regulate LMP-1 signaling

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Cytostatic effect of Epstein-Barr virus latent membrane protein-1 analyzed using tetracycline-regulated expression in B cell lines

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

BubR1 is required for a sustained mitotic spindle checkpoint arrest in human cancer cells treated with tubulin-targeting pyrrolo-1,5-benzoxazepines.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

The Epstein-Barr virus nuclear antigen-1 promotes genomic instability via induction of reactive oxygen species.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Real-time RT-PCR normalisation; strategies and considerations.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

DNA damage checkpoint control in cells exposed to ionizing radiation

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus promotes genomic instability in Burkitt's lymphoma

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growth

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus-associated Burkitt lymphomagenesis selects for downregulation of the nuclear antigen EBNA2.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Three restricted forms of Epstein-Barr virus latency counteracting apoptosis in c-myc-expressing Burkitt lymphoma cells.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus nuclear antigen 3C regulates cyclin A/p27 complexes and enhances cyclin A-dependent kinase activity

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus latent antigen 3C can mediate the degradation of the retinoblastoma protein through an SCF cellular ubiquitin ligase

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Unattached kinetochores catalyze production of an anaphase inhibitor that requires a Mad2 template to prime Cdc20 for BubR1 binding

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Expression of the Epstein-Barr virus latent membrane protein 1 induces B cell lymphoma in transgenic mice

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-barr virus-induced resistance to drugs that activate the mitotic spindle assembly checkpoint in Burkitt's lymphoma cells

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus latent membrane protein 1 represses p53-mediated DNA repair and transcriptional activity.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus latent membrane protein 1 induces micronucleus formation, represses DNA repair and enhances sensitivity to DNA-damaging agents in human epithelial cells.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor family

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Forkhead transcription factors are critical effectors of cell death and cell cycle arrest downstream of PTEN.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus nuclear antigen 2 disrupts mitotic checkpoint and causes chromosomal instability

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Use of the intracellular fluorescent dye CFSE to monitor lymphocyte migration and proliferation

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Identification of virus-encoded microRNAs

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Colonic tumorigenesis in BubR1+/-ApcMin/+ compound mutant mice is linked to premature separation of sister chromatids and enhanced genomic instability

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus nuclear antigen 3C augments Mdm2-mediated p53 ubiquitination and degradation by deubiquitinating Mdm2.

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Redefining the Epstein-Barr virus-encoded nuclear antigen EBNA-1 gene promoter and transcription initiation site in group I Burkitt lymphoma cell lines

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr nuclear antigen 1 contributes to nasopharyngeal carcinoma through disruption of PML nuclear bodies

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Persistent Epstein-Barr virus infection associated with monosomy 7 or chromosome 3 abnormality in childhood myeloproliferative disorders

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

DNA repair pathway stimulated by the forkhead transcription factor FOXO3a through the Gadd45 protein

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Latent membrane protein 2A of Epstein-Barr virus binds WW domain E3 protein-ubiquitin ligases that ubiquitinate B-cell tyrosine kinases

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Stable replication of plasmids derived from Epstein-Barr virus in various mammalian cells

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus gene expression in nasopharyngeal carcinoma

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus: 40 years on

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

31 March 2020

Epstein-Barr virus latent membrane protein (LMP1) and nuclear proteins 2 and 3C are effectors of phenotypic changes in B lymphocytes: EBNA-2 and LMP1 cooperatively induce CD23

1 reference

https://api.crossref.org/works/10.1038%2FONC.2009.258

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/ONC.2009.258

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Dimensions Publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

ResearchGate publication ID

0 references

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q39806215&oldid=2203187557"