Direct effects of Bmi1 on p53 protein stability inactivates oncoprotein stress responses in embryonal cancer precursor cells at tumor initiation. (Q39294214)

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scientific article published on 20 August 2012

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English	Direct effects of Bmi1 on p53 protein stability inactivates oncoprotein stress responses in embryonal cancer precursor cells at tumor initiation.	scientific article published on 20 August 2012

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11 February 2020

Direct effects of Bmi1 on p53 protein stability inactivates oncoprotein stress responses in embryonal cancer precursor cells at tumor initiation (English)

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11 February 2020

embryonal cancer

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based on heuristic

inferred from title

tumor initiation

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based on heuristic

inferred from title

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M Calao

1

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E O Sekyere

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H J Cui

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B B Cheung

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W D Thomas

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J Keating

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11 February 2020

J B Chen

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A Raif

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11 February 2020

K Jankowski

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11 February 2020

N P Davies

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11 February 2020

M V Bekkum

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11 February 2020

B Chen

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11 February 2020

T Ellis

14

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11 February 2020

M D Norris

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E S Kim

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J M Shohet

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11 February 2020

B J Wainwright

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H F Ding

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G M Marshall

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20 August 2012

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11 February 2020

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11 February 2020

Neuroblastoma: biological insights into a clinical enigma

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Medulloblastoma: signalling a change in treatment

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Medulloblastoma can be initiated by deletion of Patched in lineage-restricted progenitors or stem cells

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In vivo cellular and molecular mechanisms of neuronal apoptosis in the mammalian CNS.

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Mechanisms of embryonal tumor initiation: distinct roles for MycN expression and MYCN amplification.

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Patched1 deletion increases N-Myc protein stability as a mechanism of medulloblastoma initiation and progression.

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N-myc transcription molecule and oncoprotein

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The requirement for evasion of programmed cell death in neuroblastomas with MYCN amplification

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Embryonal neural tumours and cell death

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21 January 2018

p53 is a direct transcriptional target of MYCN in neuroblastoma

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https://api.crossref.org/works/10.1038%2FONC.2012.368

21 January 2018

Bmi-1 is essential for the tumorigenicity of neuroblastoma cells

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https://api.crossref.org/works/10.1038%2FONC.2012.368

21 January 2018

Mdm2 deficiency suppresses MYCN-Driven neuroblastoma tumorigenesis in vivo

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The MDM2 gene amplification database

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Novel zinc finger gene implicated as myc collaborator by retrovirally accelerated lymphomagenesis in E mu-myc transgenic mice.

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Identification of cooperating oncogenes in E mu-myc transgenic mice by provirus tagging

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Bmi-1, stem cells and cancer

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21 January 2018

Bmi1 is a MYCN target gene that regulates tumorigenesis through repression of KIF1Bbeta and TSLC1 in neuroblastoma

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Polycomb group proteins: New targets of anti-cancer therapy.

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Targeted expression of MYCN causes neuroblastoma in transgenic mice

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Cell lines from MYCN transgenic murine tumours reflect the molecular and biological characteristics of human neuroblastoma.

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Chemotherapy-induced apoptosis in a transgenic model of neuroblastoma proceeds through p53 induction

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Divorcing ARF and p53: an unsettled case

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Small molecules that reactivate p53 in renal cell carcinoma reveal a NF-kappaB-dependent mechanism of p53 suppression in tumors

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Increased frequency of aberrations in the p53/MDM2/p14(ARF) pathway in neuroblastoma cell lines established at relapse

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The E3 ubiquitin-ligase Bmi1/Ring1A controls the proteasomal degradation of Top2alpha cleavage complex - a potentially new drug target

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Structure and E3-ligase activity of the Ring-Ring complex of polycomb proteins Bmi1 and Ring1b

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Bmi1 is essential for cerebellar development and is overexpressed in human medulloblastomas

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Bcl-2 and M-Myc coexpression increases IGF-IR and features of malignant growth in neuroblastoma cell lines

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Targeted depletion of BMI1 sensitizes tumor cells to P53-mediated apoptosis in response to radiation therapy.

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shRNA knockdown of Bmi-1 reveals a critical role for p21-Rb pathway in NSC self-renewal during development

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Polyubiquitination of p53 by a ubiquitin ligase activity of p300.

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21 January 2018

Human mdm2 mediates multiple mono-ubiquitination of p53 by a mechanism requiring enzyme isomerization

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Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with a targeted deletion of the bmi-1 proto-oncogene

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Patched 1 conditional null allele in mice.

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High frequency of leukemic clones in newborn screening blood samples of children with B-precursor acute lymphoblastic leukemia

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A population-based study of the usefulness of screening for neuroblastoma

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N-myc promotes survival and induces S-phase entry of postmitotic sympathetic neurons.

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