Intestinal adenoma formation and MYC activation are regulated by cooperation between MYB and Wnt signaling. (Q39824395)

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2 February 2020

title

Intestinal adenoma formation and MYC activation are regulated by cooperation between MYB and Wnt signaling (English)

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2 February 2020

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2

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2 February 2020

Theo Mantamadiotis

9

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2 February 2020

Liang Zhao

4

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2 February 2020

Matthias Ernst

7

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2 February 2020

Shunsuke Ishii

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2 February 2020

Robert G. Ramsay

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2 February 2020

author name string

D Ciznadija

1

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2 February 2020

M L Waterman

3

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2 February 2020

D Huynh

5

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2 February 2020

R M Yu

6

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2 February 2020

T J Gonda

10

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2 February 2020

J Malaterre

12

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2 February 2020

publication date

17 July 2009

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Cell Death & Differentiation

2 February 2020

published in

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volume

16

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issue

11

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2 February 2020

page(s)

1530-1538

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https://scigraph.springernature.com/pub.10.1038/cdd.2009.94

2 February 2020

exact match

0 references

cites work

MYB function in normal and cancer cells

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c-Myb is required for progenitor cell homeostasis in colonic crypts.

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APC mutations occur early during colorectal tumorigenesis

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Identification of c-MYC as a target of the APC pathway

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The cyclin D1 gene is a target of the beta-catenin/LEF-1 pathway

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Regulation of the resident chromosomal copy of c-myc by c-Myb is involved in myeloid leukemogenesis

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Deletion of the AU-rich RNA binding protein Apobec-1 reduces intestinal tumor burden in Apc(min) mice

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Aberrant expression of an amplified c-myb oncogene in two cell lines from a colon carcinoma.

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The beta-catenin/TCF-4 complex imposes a crypt progenitor phenotype on colorectal cancer cells

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Positive autoregulation of c-myb expression via Myb binding sites in the 5' flanking region of the human c-myb gene

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Differential effects of retinoic acid on growth and apoptosis in human colon cancer cell lines associated with the induction of retinoic acid receptor beta

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Linking colorectal cancer to Wnt signaling

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The Min (multiple intestinal neoplasia) mutation: its effect on gut epithelial cell differentiation and interaction with a modifier system

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Crypt-restricted proliferation and commitment to the Paneth cell lineage following Apc loss in the mouse intestine.

21 January 2018

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Beta-catenin and TCF mediate cell positioning in the intestinal epithelium by controlling the expression of EphB/ephrinB

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c-Myc is required for the formation of intestinal crypts but dispensable for homeostasis of the adult intestinal epithelium

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Rapid loss of intestinal crypts upon conditional deletion of the Wnt/Tcf-4 target gene c-Myc

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c-Myb is critical for murine colon development

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Loss of Apc in vivo immediately perturbs Wnt signaling, differentiation, and migration

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Suppression of intestinal polyposis in Apc delta716 knockout mice by inhibition of cyclooxygenase 2 (COX-2)

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An expression-based site of origin diagnostic method designed for clinical application to cancer of unknown origin

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Mutations in the MYB intron I regulatory sequence increase transcription in colon cancers

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Quantitative analysis of the hormone-induced hyperacetylation of histone H3 associated with the steroidogenic acute regulatory protein gene promoter

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Biological characterization and oncogene expression in human colorectal carcinoma cell lines

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7 January 2021

Role of c-Myc in intestinal tumorigenesis of the ApcMin/+ mouse

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7 January 2021

Beta-catenin regulates expression of cyclooxygenase-2 in articular chondrocytes

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7 January 2021

Elevated cyclooxygenase-2 levels in Min mouse adenomas

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7 January 2021