IL-17A plays a central role in the expression of psoriasis signature genes through the induction of IκB-ζ in keratinocytes. (Q39947458)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26944069%20AND%20SRC:MED&resulttype=core&format=json

9 March 2020

title

IL-17A plays a central role in the expression of psoriasis signature genes through the induction of IκB-ζ in keratinocytes (English)

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9 March 2020

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1

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9 March 2020

author name string

Toru Hirao

2

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9 March 2020

Keisuke Tawa

3

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9 March 2020

Koki Hirashima

4

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9 March 2020

Shigeyuki Kon

5

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9 March 2020

Yuichi Kitai

6

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9 March 2020

Tadashi Matsuda

7

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9 March 2020

publication date

3 March 2016

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9 March 2020

published in

International Immunology

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9 March 2020

volume

28

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9 March 2020

issue

9

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9 March 2020

page(s)

443-452

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9 March 2020

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

21 January 2018

Immunology of psoriasis

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

21 January 2018

Cytokines in psoriasis

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Interleukin 17A: toward a new understanding of psoriasis pathogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Cellular sources of IL-17 in psoriasis: a paradigm shift?

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Th17 cytokines interleukin (IL)-17 and IL-22 modulate distinct inflammatory and keratinocyte-response pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Integrative responses to IL-17 and TNF-α in human keratinocytes account for key inflammatory pathogenic circuits in psoriasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Interleukin-17 and its expanding biological functions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Effective treatment of psoriasis with etanercept is linked to suppression of IL-17 signaling, not immediate response TNF genes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Detection of interferon-gamma mRNA in psoriatic epidermis by polymerase chain reaction.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

The Cytokine Network in Lesional and Lesion-Free Psoriatic Skin Is Characterized by a T-Helper Type 1 Cell-Mediated Response

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Biology of interleukin-22.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IL-17A is essential for cell activation and inflammatory gene circuits in subjects with psoriasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IL-17 induces an expanded range of downstream genes in reconstituted human epidermis model

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IL-17A and IFN-γ synergistically induce RNase 7 expression via STAT3 in primary keratinocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Jun activation domain-binding protein 1 (JAB1) is required for the optimal response to interferons.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Transcriptome analysis of psoriasis in a large case-control sample: RNA-seq provides insights into disease mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Expanding the psoriasis disease profile: interrogation of the skin and serum of patients with moderate-to-severe psoriasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IL-19 is a component of the pathogenetic IL-23/IL-17 cascade in psoriasis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Clinical improvement in psoriasis with specific targeting of interleukin-23.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Guselkumab (an IL-23-specific mAb) demonstrates clinical and molecular response in patients with moderate-to-severe psoriasis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Tildrakizumab (MK-3222), an anti-interleukin-23p19 monoclonal antibody, improves psoriasis in a phase IIb randomized placebo-controlled trial

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Chemokines and other mediators as therapeutic targets in psoriasis vulgaris

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Structural organization and regulation of the small proline-rich family of cornified envelope precursors suggest a role in adaptive barrier function

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Beta-defensin-2 protein is a serum biomarker for disease activity in psoriasis and reaches biologically relevant concentrations in lesional skin

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Requirements for two proximal NF-kappaB binding sites and IkappaB-zeta in IL-17A-induced human beta-defensin 2 expression by conducting airway epithelium

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Induction of neutrophil gelatinase-associated lipocalin expression by co-stimulation with interleukin-17 and tumor necrosis factor-alpha is controlled by IkappaB-zeta but neither by C/EBP-beta nor C/EBP-delta.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IκBζ is a key driver in the development of psoriasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Enhanced apoptosis by disruption of the STAT3-IκB-ζ signaling pathway in epithelial cells induces Sjögren's syndrome-like autoimmune disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IκB-ζ controls the constitutive NF-κB target gene network and survival of ABC DLBCL

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Regulation of tumour necrosis factor signalling: live or let die

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

Enhanced meta-analysis and replication studies identify five new psoriasis susceptibility loci

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

IκBζ regulates TH17 development by cooperating with ROR nuclear receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FINTIMM%2FDXW011

21 January 2018

Identifiers

DOI

10.1093/INTIMM/DXW011

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26944069%20AND%20SRC:MED&resulttype=core&format=json

9 March 2020

1 reference