Biochemical characterisation of TCTP questions its function as a guanine nucleotide exchange factor for Rheb (Q39952074)

15 September 2017

title

Biochemical characterisation of TCTP questions its function as a guanine nucleotide exchange factor for Rheb (English)

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Translationally controlled tumor protein Dmel_CG4800

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main subject

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GOA release 2020-03-11

M Schwarten

4

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author name string

H Rehmann

1

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15 September 2017

M Brüning

2

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C Berghaus

3

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K Köhler

5

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H Stocker

6

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R Stoll

7

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F J Zwartkruis

8

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A Wittinghofer

9

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language of work or name

English

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publication date

8 August 2008

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15 September 2017

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15 September 2017

volume

582

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issue

20

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page(s)

3005-3010

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Rheb is a direct target of the tuberous sclerosis tumour suppressor proteins

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cites work

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Drosophila TCTP is essential for growth and proliferation through regulation of dRheb GTPase

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Structure of TCTP reveals unexpected relationship with guanine nucleotide-free chaperones

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Characterization of the activation of the Rap-specific exchange factor Epac by cyclic nucleotides.

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Insulin activation of Rheb, a mediator of mTOR/S6K/4E-BP signaling, is inhibited by TSC1 and 2

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Sequence-specific 1H, 13C, and 15N backbone assignment of the activated 21 kDa GTPase rRheb.

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Nucleotide exchange via local protein unfolding--structure of Rab8 in complex with MSS4.

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A knockout mouse approach reveals that TCTP functions as an essential factor for cell proliferation and survival in a tissue- or cell type-specific manner

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gigas, a Drosophila homolog of tuberous sclerosis gene product-2, regulates the cell cycle

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The Drosophila tuberous sclerosis complex gene homologs restrict cell growth and cell proliferation

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Negative regulation of TSC1-TSC2 by mammalian D-type cyclins.

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Inactivation of the cyclin-dependent kinase inhibitor p27 upon loss of the tuberous sclerosis complex gene-2

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Re-evaluating the roles of proposed modulators of mammalian target of rapamycin complex 1 (mTORC1) signaling

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NMR Analysis of Interacting Soluble Forms of the Cell−Cell Recognition Molecules CD2 and CD48†

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Identifiers

DOI

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15 September 2017

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