The nucleoside analog sangivamycin induces apoptotic cell death in breast carcinoma MCF7/adriamycin-resistant cells via protein kinase Cdelta and JNK activation (Q40156976)

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scientific article published on 19 March 2007

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English	The nucleoside analog sangivamycin induces apoptotic cell death in breast carcinoma MCF7/adriamycin-resistant cells via protein kinase Cdelta and JNK activation	scientific article published on 19 March 2007

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scholarly article

1 reference

Europe PubMed Central

18 September 2017

The nucleoside analog sangivamycin induces apoptotic cell death in breast carcinoma MCF7/adriamycin-resistant cells via protein kinase Cdelta and JNK activation (English)

1 reference

Europe PubMed Central

18 September 2017

apoptotic process

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breast carcinoma

1 reference

based on heuristic

inferred from title

author name string

Sung A Lee

1

1 reference

Europe PubMed Central

18 September 2017

Mira Jung

2

1 reference

Europe PubMed Central

18 September 2017

language of work or name

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publication date

19 March 2007

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Europe PubMed Central

18 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

18 September 2017

282

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Europe PubMed Central

18 September 2017

20

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Europe PubMed Central

18 September 2017

15271-15283

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Europe PubMed Central

18 September 2017

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The proto-oncogene Bcl-2 and its role in regulating apoptosis

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Protein kinase C involvement in apoptosis

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Jun and JNK kinase are activated in thymocytes in response to VM26 and radiation but not glucocorticoids.

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JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

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Transcription factor ATF2 regulation by the JNK signal transduction pathway

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BCL-2 is phosphorylated and inactivated by an ASK1/Jun N-terminal protein kinase pathway normally activated at G(2)/M.

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JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis

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Role of ATM in oxidative stress-mediated c-Jun phosphorylation in response to ionizing radiation and CdCl2.

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

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Requirement for p53 and p21 to sustain G2 arrest after DNA damage

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Hypoxia-induced apoptosis in human cells with normal p53 status and function, without any alteration in the nuclear protein level.

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Expression of the mutated p53 tumor suppressor protein and its molecular and biochemical characterization in multidrug resistant MCF-7/Adr human breast cancer cells

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Resistance of MCF7 human breast carcinoma cells to TNF-induced cell death is associated with loss of p53 function

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The CED-3/ICE-like protease Mch2 is activated during apoptosis and cleaves the death substrate lamin A

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ICE-LAP3, a novel mammalian homologue of the Caenorhabditis elegans cell death protein Ced-3 is activated during Fas- and tumor necrosis factor-induced apoptosis

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Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain

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Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

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c-Jun N-terminal kinase (JNK)-mediated modulation of brain mitochondria function: new target proteins for JNK signalling in mitochondrion-dependent apoptosis

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ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

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10.1074/JBC.M701362200

1 reference

Europe PubMed Central

18 September 2017

1 reference

Europe PubMed Central

18 September 2017

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