c-Jun promotes cellular survival by suppression of PTEN. (Q40283457)

19 September 2017

title

c-Jun promotes cellular survival by suppression of PTEN. (English)

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19 September 2017

K Hettinger

1

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19 September 2017

F Vikhanskaya

2

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19 September 2017

M K Poh

3

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19 September 2017

M K Lee

4

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19 September 2017

I de Belle

5

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19 September 2017

J-T Zhang

6

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19 September 2017

S A G Reddy

7

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19 September 2017

K Sabapathy

8

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19 September 2017

publication date

5 May 2006

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Cell Death & Differentiation

19 September 2017

published in

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19 September 2017

volume

14

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19 September 2017

issue

2

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19 September 2017

page(s)

218-229

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401946

19 September 2017

exact match

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Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

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Distinct roles for JNK1 and JNK2 in regulating JNK activity and c-Jun-dependent cell proliferation

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AP-1 blockade in breast cancer cells causes cell cycle arrest by suppressing G1 cyclin expression and reducing cyclin-dependent kinase activity.

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Control of cell cycle progression by c-Jun is p53 dependent

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c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms

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JNK1 modulates osteoclastogenesis through both c-Jun phosphorylation-dependent and -independent mechanisms

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Liver tumor development. c-Jun antagonizes the proapoptotic activity of p53

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c-Jun-dependent CD95-L expression is a rate-limiting step in the induction of apoptosis by alkylating agents

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The AP-1 transcription factor c-Jun is required for efficient axonal regeneration

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JNK2 is required for efficient T-cell activation and apoptosis but not for normal lymphocyte development.

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Defective neural tube morphogenesis and altered apoptosis in the absence of both JNK1 and JNK2.

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Phosphorylation of c-Jun is necessary for apoptosis induced by survival signal withdrawal in cerebellar granule neurons.

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Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

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PTEN modulates cell cycle progression and cell survival by regulating phosphatidylinositol 3,4,5,-trisphosphate and Akt/protein kinase B signaling pathway

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Regulation of PTEN transcription by p53

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The Egr-1 transcription factor directly activates PTEN during irradiation-induced signalling

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Regulation of constitutive expression of mouse PTEN by the 5'-untranslated region

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Identifiers

DOI

10.1038/SJ.CDD.4401946

1 reference

Dimensions Publication ID

19 September 2017

1018011158

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1 reference