Prostate epithelial cells can act as early sensors of infection by up-regulating TLR4 expression and proinflammatory mediators upon LPS stimulation. (Q40307735)

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scientific article published on 7 March 2006

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English	Prostate epithelial cells can act as early sensors of infection by up-regulating TLR4 expression and proinflammatory mediators upon LPS stimulation.	scientific article published on 7 March 2006

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scholarly article

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17 January 2020

Prostate epithelial cells can act as early sensors of infection by up-regulating TLR4 expression and proinflammatory mediators upon LPS stimulation (English)

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17 January 2020

toll-like receptor

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Ruben D. Motrich

3

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17 January 2020

author name string

Gerardo Gatti

1

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17 January 2020

Virginia Rivero

2

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17 January 2020

Mariana Maccioni

4

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17 January 2020

publication date

7 March 2006

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17 January 2020

Journal of Leukocyte Biology

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17 January 2020

79

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17 January 2020

5

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17 January 2020

989-998

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17 January 2020

Toll-like receptor 4-mediated signaling by epithelial surfaces: necessity or threat?

1 reference

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Toll-like receptors: critical proteins linking innate and acquired immunity

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Decreased expression of Toll-like receptor-4 and MD-2 correlates with intestinal epithelial cell protection against dysregulated proinflammatory gene expression in response to bacterial lipopolysaccharide

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TLR4 and MD-2 expression is regulated by immune-mediated signals in human intestinal epithelial cells

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Commensal-associated molecular patterns induce selective toll-like receptor-trafficking from apical membrane to cytoplasmic compartments in polarized intestinal epithelium

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Toll-like receptor 4 resides in the Golgi apparatus and colocalizes with internalized lipopolysaccharide in intestinal epithelial cells

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Intracellular Recognition of Lipopolysaccharide by Toll-like Receptor 4 in Intestinal Epithelial Cells

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