Raised expression of the antiapoptotic protein ped/pea-15 increases susceptibility to chemically induced skin tumor development. (Q40393314)

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4 January 2020

title

Raised expression of the antiapoptotic protein ped/pea-15 increases susceptibility to chemically induced skin tumor development (English)

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4 January 2020

1

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4 January 2020

Gregoryalexander Raciti

6

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4 January 2020

author name string

Giuseppe Perruolo

2

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4 January 2020

Silvana Libertini

3

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4 January 2020

Stefania Santopietro

4

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4 January 2020

Giancarlo Troncone

5

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4 January 2020

Francesco Oriente

7

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4 January 2020

Giuseppe Portella

8

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4 January 2020

Claudia Miele

9

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4 January 2020

Francesco Beguinot

10

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4 January 2020

language of work or name

English

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publication date

1 October 2005

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4 January 2020

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4 January 2020

volume

24

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4 January 2020

issue

47

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4 January 2020

page(s)

7012-7021

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Analysis of APAF-1 expression in human cutaneous melanoma progression

4 January 2020

cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Carcinogenesis in mouse and human cells: parallels and paradoxes

21 January 2018

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Overweight, obesity and cancer: epidemiological evidence and proposed mechanisms

21 January 2018

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Multiple members of the mitogen-activated protein kinase family are necessary for PED/PEA-15 anti-apoptotic function

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

PED/PEA-15 gene controls glucose transport and is overexpressed in type 2 diabetes mellitus

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

PED/PEA-15: an anti-apoptotic molecule that regulates FAS/TNFR1-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Death receptors in chemotherapy and cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

PEA-15 mediates cytoplasmic sequestration of ERK MAP kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

PEA-15 is inhibited by adenovirus E1A and plays a role in ERK nuclear export and Ras-induced senescence

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

AGAR SUSPENSION CULTURE FOR THE SELECTIVE ASSAY OF CELLS TRANSFORMED BY POLYOMA VIRUS.

21 January 2018

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Death effector domain protein PEA-15 potentiates Ras activation of extracellular signal receptor-activated kinase by an adhesion-independent mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Omi/HtrA2 promotes cell death by binding and degrading the anti-apoptotic protein ped/pea-15

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Protein kinase B/Akt binds and phosphorylates PED/PEA-15, stabilizing its antiapoptotic action

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Expression of MAT1/PEA-15 mRNA isoforms during physiological and neoplastic changes in the mouse mammary gland.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

RSK2 activity is regulated by its interaction with PEA-15

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

Overexpression of the ped/pea-15 gene causes diabetes by impairing glucose-stimulated insulin secretion in addition to insulin action

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208871

21 January 2018

Identifiers

DOI

10.1038/SJ.ONC.1208871

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:16044159%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

4 January 2020

1042808960

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1 reference