The antiapoptotic effect of fibroblast growth factor-2 is mediated through nuclear factor-kappaB activation induced via interaction between Akt and IkappaB kinase-beta in breast cancer cells. (Q40430306)

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English	The antiapoptotic effect of fibroblast growth factor-2 is mediated through nuclear factor-kappaB activation induced via interaction between Akt and IkappaB kinase-beta in breast cancer cells.	scientific article

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11 January 2020

The antiapoptotic effect of fibroblast growth factor-2 is mediated through nuclear factor-kappaB activation induced via interaction between Akt and IkappaB kinase-beta in breast cancer cells (English)

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11 January 2020

Franck Vandermoere

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11 January 2020

Jérôme Lemoine

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11 January 2020

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Ikram El Yazidi-Belkoura

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11 January 2020

Eric Adriaenssens

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11 January 2020

Hubert Hondermarck

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1 August 2005

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11 January 2020

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11 January 2020

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11 January 2020

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11 January 2020

5482-5491

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11 January 2020

https://scigraph.springernature.com/pub.10.1038/sj.onc.1208713

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Antiapoptotic signalling by the insulin-like growth factor I receptor, phosphatidylinositol 3-kinase, and Akt.

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Fibroblast growth factor-2 induces translational regulation of Bcl-XL and Bcl-2 via a MEK-dependent pathway: correlation with resistance to etoposide-induced apoptosis

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Interaction of heparan sulfate from mammary cells with acidic fibroblast growth factor (FGF) and basic FGF. Regulation of the activity of basic FGF by high and low affinity binding sites in heparan sulfate

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NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

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The mitogenic signaling pathway for fibroblast growth factor-2 involves the tyrosine phosphorylation of cyclin D2 in MCF-7 human breast cancer cells

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The p75 neurotrophin receptor (p75NTR) alters tumor necrosis factor-mediated NF-kappaB activity under physiological conditions, but direct p75NTR-mediated NF-kappaB activation requires cell stress

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7 January 2021

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The proliferative and migratory activities of breast cancer cells can be differentially regulated by heparan sulfates

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A signaling pathway leading to metastasis is controlled by N-cadherin and the FGF receptor

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Identifiers

10.1038/SJ.ONC.1208713

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11 January 2020

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11 January 2020

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