Role of poly(ADP-ribose) polymerase activity in imatinib mesylate-induced cell death (Q40437209)

20 September 2017

title

Role of poly(ADP-ribose) polymerase activity in imatinib mesylate-induced cell death (English)

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20 September 2017

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Moehring A

1

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20 September 2017

Wohlbold L

series ordinal

2

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20 September 2017

Aulitzky WE

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3

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20 September 2017

van der Kuip H

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4

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20 September 2017

publication date

1 June 2005

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Cell Death & Differentiation

20 September 2017

published in

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20 September 2017

volume

12

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20 September 2017

issue

6

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20 September 2017

page(s)

627-636

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The BCR-ABL oncogene transforms Rat-1 cells and cooperates with v-myc

20 September 2017

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Transformation of an interleukin 3-dependent hematopoietic cell line by the chronic myelogenous leukemia-specific P210bcr/abl protein

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Bcr/Abl expression stimulates integrin function in hematopoietic cell lines

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Regulation of Bcl-2 gene expression by BCR-ABL is mediated by Ras.

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Adhesion to fibronectin selectively protects Bcr-Abl+ cells from DNA damage-induced apoptosis

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Imatinib mesylate (STI571) prevents the mutator phenotype of Bcr-Abl in hematopoietic cell lines

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Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells

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A novel mechanism for imatinib mesylate-induced cell death of BCR-ABL-positive human leukemic cells: caspase-independent, necrosis-like programmed cell death mediated by serine protease activity

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Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

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Mediation of poly(ADP-ribose) polymerase-1-dependent cell death by apoptosis-inducing factor

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Molecular characterization of mitochondrial apoptosis-inducing factor

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DNA-binding properties of poly(ADP-ribose) polymerase: a target for anticancer therapy

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PARP goes transcription

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Recombinant IFN-alpha2b treatment activates poly (ADPR) polymerase-1 (PARP-1) in KB cancer cells

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Poly(ADP-ribosyl)ation inhibitors: promising drug candidates for a wide variety of pathophysiologic conditions

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BCR-ABL gene mutations in relation to clinical resistance of Philadelphia-chromosome-positive leukaemia to STI571: a prospective study

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Transformation of hematopoietic cells by BCR/ABL requires activation of a PI-3k/Akt-dependent pathway.

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7 January 2021

Role of ERK activation in growth and erythroid differentiation of K562 cells

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7 January 2021

Physiology and pathophysiology of poly(ADP-ribosyl)ation

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7 January 2021

Poly(ADP-ribose) polymerase-1-mediated cell death in astrocytes requires NAD+ depletion and mitochondrial permeability transition

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7 January 2021

Effects of the Bcr/abl kinase inhibitors STI571 and adaphostin (NSC 680410) on chronic myelogenous leukemia cells in vitro

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7 January 2021

Nuclear and mitochondrial conversations in cell death: PARP-1 and AIF signaling

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7 January 2021

Mitochondrial release of AIF and EndoG requires caspase activation downstream of Bax/Bak-mediated permeabilization

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Mitochondria, AIF and caspases--rivaling for cell death execution.

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7 January 2021

Enhanced phosphorylation of p53 by ATM in response to DNA damage

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7 January 2021

Phosphatidylinositol 3-kinase inhibitors, Wortmannin or LY294002, inhibited accumulation of p21 protein after gamma-irradiation by stabilization of the protein

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7 January 2021

Molecular cloning of the akt oncogene and its human homologues AKT1 and AKT2: amplification of AKT1 in a primary human gastric adenocarcinoma

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7 January 2021

The phosphatidylinositol 3-Kinase AKT pathway in human cancer

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7 January 2021

The phosphoinositide 3-kinase pathway

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401608

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Dimensions Publication ID

20 September 2017

1028815640

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