Rapamycin induces the fusion-type independent downregulation of the EWS/FLI-1 proteins and inhibits Ewing's sarcoma cell proliferation (Q40606312)

22 September 2017

Rapamycin induces the fusion-type independent downregulation of the EWS/FLI-1 proteins and inhibits Ewing's sarcoma cell proliferation (English)

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22 September 2017

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Silvia Mateo-Lozano

1

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22 September 2017

Oscar M Tirado

series ordinal

2

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22 September 2017

Vicente Notario

series ordinal

3

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22 September 2017

language of work or name

English

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publication date

1 December 2003

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22 September 2017

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22 September 2017

22

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22 September 2017

58

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22 September 2017

9282-9287

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1207081

22 September 2017

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Variability in gene expression patterns of Ewing tumor cell lines differing in EWS-FLI1 fusion type

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Association of EWS-FLI1 type 1 fusion with lower proliferative rate in Ewing's sarcoma

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Rapamycin-induced G1 arrest in cycling B-CLL cells is associated with reduced expression of cyclin D3, cyclin E, cyclin A, and survivin

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Gene fusion with an ETS DNA-binding domain caused by chromosome translocation in human tumours

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Loss of p16 pathways stabilizes EWS/FLI1 expression and complements EWS/FLI1 mediated transformation.

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Regulation of translation initiation by FRAP/mTOR

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Rapamycin is an effective inhibitor of human renal cancer metastasis.

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Downregulation and forced expression of EWS-Fli1 fusion gene results in changes in the expression of G(1)regulatory genes.

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Identification of p21WAF1/CIP1 as a direct target of EWS-Fli1 oncogenic fusion protein

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Treatment of Ewing sarcoma family of tumors: current status and outlook for the future

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Cyclin-dependent kinases: initial approaches to exploit a novel therapeutic target

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FRAP-p70s6K signaling is required for pancreatic cancer cell proliferation

21 January 2018

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Beta-platelet-derived growth factor receptor mediates motility and growth of Ewing's sarcoma cells.

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Rapamycin induces heme oxygenase-1 in human pulmonary vascular cells: implications in the antiproliferative response to rapamycin

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PDGF-C is an EWS/FLI induced transforming growth factor in Ewing family tumors

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p70s6k function is essential for G1 progression

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A link between basic fibroblast growth factor (bFGF) and EWS/FLI-1 in Ewing's sarcoma cells

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7 January 2021

Repression of the gene encoding the TGF-beta type II receptor is a major target of the EWS-FLI1 oncoprotein

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7 January 2021

Molecular genetics of chromosome translocations involving EWS and related family members

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Combinatorial generation of variable fusion proteins in the Ewing family of tumours.

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1207081

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Dimensions Publication ID

22 September 2017

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1 reference