Inhibition of PKCalpha induces a PKCdelta-dependent apoptotic program in salivary epithelial cells. (Q40654556)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12700627%20AND%20SRC:MED&resulttype=core&format=json

2 November 2019

title

Inhibition of PKCalpha induces a PKCdelta-dependent apoptotic program in salivary epithelial cells (English)

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2 November 2019

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4

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2 November 2019

author name string

Matassa AA

1

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2 November 2019

Kalkofen RL

2

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2 November 2019

Carpenter L

3

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2 November 2019

Reyland ME

5

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2 November 2019

publication date

1 March 2003

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Cell Death & Differentiation

2 November 2019

published in

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2 November 2019

volume

10

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2 November 2019

issue

3

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2 November 2019

page(s)

269-277

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401149

2 November 2019

exact match

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PKCdelta is required for mitochondrial-dependent apoptosis in salivary epithelial cells.

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Protein kinase C-alpha overexpression stimulates Akt activity and suppresses apoptosis induced by interleukin 3 withdrawal

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Activation of stress-activated protein kinase/c-Jun NH2-terminal kinase and p38 kinase in calphostin C-induced apoptosis requires caspase-3-like proteases but is dispensable for cell death.

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Activation of PKCalpha downstream from caspases during apoptosis induced by 7-hydroxystaurosporine or the topoisomerase inhibitors, camptothecin and etoposide, in human myeloid leukemia HL60 cells

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The role of c-Jun N-terminal kinase (JNK) in apoptosis induced by ultraviolet C and gamma radiation. Duration of JNK activation may determine cell death and proliferation

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c-Jun NH2-terminal kinase regulation of the apoptotic response of small cell lung cancer cells to ultraviolet radiation

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401149

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12700627%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

2 November 2019

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