Target-derived GFRalpha1 as an attractive guidance signal for developing sensory and sympathetic axons via activation of Cdk5. (Q40693379)

22 September 2017

title

Target-derived GFRalpha1 as an attractive guidance signal for developing sensory and sympathetic axons via activation of Cdk5. (English)

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22 September 2017

Fernanda Ledda

1

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22 September 2017

Gustavo Paratcha

series ordinal

2

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22 September 2017

Carlos F Ibáñez

series ordinal

3

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22 September 2017

language of work or name

English

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publication date

1 October 2002

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22 September 2017

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22 September 2017

volume

36

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22 September 2017

issue

3

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22 September 2017

page(s)

387-401

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The GDNF family: signalling, biological functions and therapeutic value

22 September 2017

cites work

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GDNF is an age-specific survival factor for sensory and autonomic neurons

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GFRalpha1 is an essential receptor component for GDNF in the developing nervous system and kidney

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The cyclin-dependent kinase Cdk5 controls multiple aspects of axon patterning in vivo

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GDNF signalling through the Ret receptor tyrosine kinase

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GFR alpha1-deficient mice have deficits in the enteric nervous system and kidneys.

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Brain-derived neurotrophic factor and glial cell line-derived neurotrophic factor are required simultaneously for survival of dopaminergic primary sensory neurons in vivo.

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ERK induces p35, a neuron-specific activator of Cdk5, through induction of Egr1.

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GDNF: a potent survival factor for motoneurons present in peripheral nerve and muscle

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GDNF-induced activation of the ret protein tyrosine kinase is mediated by GDNFR-alpha, a novel receptor for GDNF

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A GPI-linked protein that interacts with Ret to form a candidate neurturin receptor

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Renal and neuronal abnormalities in mice lacking GDNF

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Glial cell line-derived neurotrophic factor promotes the survival and morphologic differentiation of Purkinje cells

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Differential regulation of mRNAs for GDNF and its receptors Ret and GDNFR alpha after sciatic nerve lesion in the mouse

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The cdk5/p35 kinase is essential for neurite outgrowth during neuronal differentiation

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Glial cell line-derived neurotrophic factor-dependent RET activation can be mediated by two different cell-surface accessory proteins

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GFRalpha-mediated localization of RET to lipid rafts is required for effective downstream signaling, differentiation, and neuronal survival

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Complementary and overlapping expression of glial cell line-derived neurotrophic factor (GDNF), c-ret proto-oncogene, and GDNF receptor-alpha indicates multiple mechanisms of trophic actions in the adult rat CNS.

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Distinct regulatory pathways control neurofilament expression and neurotransmitter synthesis in immortalized serotonergic neurons

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Rap1 mediates sustained MAP kinase activation induced by nerve growth factor

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Identifiers

DOI

1 reference

22 September 2017

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