Increased ROS levels contribute to elevated transcription factor and MAP kinase activities in malignantly progressed mouse keratinocyte cell lines (Q40920084)

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English	Increased ROS levels contribute to elevated transcription factor and MAP kinase activities in malignantly progressed mouse keratinocyte cell lines	scientific article

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scholarly article

1 reference

Europe PubMed Central

24 September 2017

Increased ROS levels contribute to elevated transcription factor and MAP kinase activities in malignantly progressed mouse keratinocyte cell lines (English)

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Europe PubMed Central

24 September 2017

1 reference

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Gupta A

1

1 reference

Europe PubMed Central

24 September 2017

Rosenberger SF

2

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Europe PubMed Central

24 September 2017

Bowden GT

3

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Europe PubMed Central

24 September 2017

language of work or name

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publication date

1 November 1999

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Europe PubMed Central

24 September 2017

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Europe PubMed Central

24 September 2017

20

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Europe PubMed Central

24 September 2017

11

1 reference

Europe PubMed Central

24 September 2017

2063-2073

1 reference

Europe PubMed Central

24 September 2017

Multistage carcinogenesis in mouse skin

1 reference

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Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells

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Constitutive AP-1 DNA binding and transactivating ability of malignant but not benign mouse epidermal cells

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A dominant negative mutant of jun blocking 12-O-tetradecanoylphorbol-13-acetate-induced invasion in mouse keratinocytes

1 reference

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21 January 2018

AP-1 function and regulation

1 reference

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c-jun and multistage carcinogenesis: association of overexpression of introduced c-jun with progression toward a neoplastic endpoint in mouse JB6 cells sensitive to tumor promoter-induced transformation

1 reference

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AP1/jun function is differentially induced in promotion-sensitive and resistant JB6 cells.

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NF-kappa B: ten years after

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Expression of dominant negative Jun inhibits elevated AP-1 and NF-kappaB transactivation and suppresses anchorage independent growth of HPV immortalized human keratinocytes

1 reference

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Redox regulation of NF-kappa B activation

1 reference

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Mitogen-activated protein kinase cascades and regulation of gene expression

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Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

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The cellular response to oxidative stress: influences of mitogen-activated protein kinase signalling pathways on cell survival

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The regulation of AP-1 activity by mitogen-activated protein kinases

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Activation of the IkappaB alpha kinase complex by MEKK1, a kinase of the JNK pathway

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Activation of p38 mitogen-activated protein kinase by sodium salicylate leads to inhibition of tumor necrosis factor-induced IkappaB alpha phosphorylation and degradation

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Activation of mitogen-activated protein kinase by H2O2. Role in cell survival following oxidant injury

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Different role of lipid peroxidation in oxidative stress-induced lethal injury in normal and tumor thymocytes.

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The role of oxygen radicals as a possible mechanism of tumor promotion

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Role of free radicals in the initiation and promotion of radiation transformation in vitro.

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Manganese superoxide dismutase expression inhibits soft agar growth in JB6 clone41 mouse epidermal cells

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Mitogenic signaling mediated by oxidants in Ras-transformed fibroblasts

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The pathogenesis of squamous cell cancer: lessons learned from studies of skin carcinogenesis.

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Molecular mechanisms of ultraviolet radiation carcinogenesis

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Extracellular signal-regulated kinase 1/2-mediated phosphorylation of JunD and FosB is required for okadaic acid-induced activator protein 1 activation

1 reference

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Progressive abrogation of TGF-beta 1 and EGF growth control is associated with tumour progression in ras-transfected human keratinocytes

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

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Active oxygen induced DNA strand breakage and poly ADP-ribosylation in promotable and non-promotable JB6 mouse epidermal cells.

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A protein kinase involved in the regulation of inflammatory cytokine biosynthesis

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Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

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The NF-kappa B and I kappa B proteins: new discoveries and insights

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p38 and extracellular signal-regulated kinase mitogen-activated protein kinase pathways are required for nuclear factor-kappaB p65 transactivation mediated by tumor necrosis factor.

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Control of transcription factors by signal transduction pathways: the beginning of the end

1 reference

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Binding of promoter-associated AP-1 is not altered during induction and subsequent repression of the c-jun promoter by TPA and UV irradiation.

1 reference

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Activation of stress-activated MAP protein kinases up-regulates expression of transgenes driven by the cytomegalovirus immediate/early promoter

1 reference

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Shortage of mitogen-activated protein kinase is responsible for resistance to AP-1 transactivation and transformation in mouse JB6 cells.

1 reference

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Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

1 reference

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How MAP kinases are regulated

1 reference

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Sodium salicylate induces apoptosis via p38 mitogen-activated protein kinase but inhibits tumor necrosis factor-induced c-Jun N-terminal kinase/stress-activated protein kinase activation.

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p38 Mitogen-activated protein kinase is a critical component of the redox-sensitive signaling pathways activated by angiotensin II. Role in vascular smooth muscle cell hypertrophy.

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Activation of stress-activated protein kinase-3 (SAPK3) by cytokines and cellular stresses is mediated via SAPKK3 (MKK6); comparison of the specificities of SAPK3 and SAPK2 (RK/p38)

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A novel kinase cascade triggered by stress and heat shock that stimulates MAPKAP kinase-2 and phosphorylation of the small heat shock proteins

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Induction of apoptosis by SB202190 through inhibition of p38beta mitogen-activated protein kinase.

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The level of intracellular glutathione is a key regulator for the induction of stress-activated signal transduction pathways including Jun N-terminal protein kinases and p38 kinase by alkylating agents

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Multiple Ras functions can contribute to mammalian cell transformation

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Redox regulation of fos and jun DNA-binding activity in vitro

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Inhibition of MAP Kinase Kinase Blocks the Differentiation of PC-12 Cells Induced by Nerve Growth Factor

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Malignant conversion of mouse skin tumours is increased by tumour initiators and unaffected by tumour promoters

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Identifiers

10.1093/CARCIN/20.11.2063

1 reference

Europe PubMed Central

24 September 2017

1 reference

Europe PubMed Central

24 September 2017

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