Circulating high-molecular-weight RAGE ligands activate pathways implicated in the development of diabetic nephropathy (Q42171678)

17 October 2017

Circulating high-molecular-weight RAGE ligands activate pathways implicated in the development of diabetic nephropathy (English)

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17 October 2017

Piyush M Srivastava

4

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Sheila K Patel

Sheila K Patel

3

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Josephine Forbes

Josephine M Forbes

13

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Merlin Thomas

Merlin C Thomas

8

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Louise M Burrell

11

Louise M Burrell

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17 October 2017

Sally A Penfold

1

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17 October 2017

Melinda T Coughlan

2

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17 October 2017

Karly C Sourris

5

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17 October 2017

David Steer

6

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17 October 2017

Diane E Webster

7

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17 October 2017

Richard J MacIsaac

9

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17 October 2017

George Jerums

10

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17 October 2017

Mark E Cooper

12

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17 October 2017

language of work or name

English

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publication date

12 May 2010

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17 October 2017

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17 October 2017

78

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17 October 2017

287-295

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17 October 2017

3

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https://scigraph.springernature.com/pub.10.1038/ki.2010.134

17 October 2017

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cites work

Retardation by Aminoguanidine of Development of Albuminuria, Mesangial Expansion, and Tissue Fluorescence in Streptozocin-Induced Diabetic Rat

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https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Role of oxidative stress in diabetic complications: a new perspective on an old paradigm

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https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Increased accumulation of the glycoxidation product N(epsilon)-(carboxymethyl)lysine in human tissues in diabetes and aging.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

N(epsilon)-(carboxymethyl)lysine levels in patients with type 2 diabetes: role of renal function

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Skin collagen glycation, glycoxidation, and crosslinking are lower in subjects with long-term intensive versus conventional therapy of type 1 diabetes: relevance of glycated collagen products versus HbA1c as markers of diabetic complications. DCCT S

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Reduction of the accumulation of advanced glycation end products by ACE inhibition in experimental diabetic nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Renoprotective effects of a novel inhibitor of advanced glycation

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https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

The breakdown of preexisting advanced glycation end products is associated with reduced renal fibrosis in experimental diabetes.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

RAGE mediates amyloid-beta peptide transport across the blood-brain barrier and accumulation in brain

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https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Understanding RAGE, the receptor for advanced glycation end products

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Toll-like receptor 9-dependent activation by DNA-containing immune complexes is mediated by HMGB1 and RAGE

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

RAGE-induced cytosolic ROS promote mitochondrial superoxide generation in diabetes.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

RAGE drives the development of glomerulosclerosis and implicates podocyte activation in the pathogenesis of diabetic nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Modulation of soluble receptor for advanced glycation end products by angiotensin-converting enzyme-1 inhibition in diabetic nephropathy.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Soluble RAGE but not endogenous secretory RAGE is associated with albuminuria in patients with type 2 diabetes

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Association between serum levels of soluble receptor for advanced glycation end products and circulating advanced glycation end products in type 2 diabetes

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Hexameric calgranulin C (S100A12) binds to the receptor for advanced glycated end products (RAGE) using symmetric hydrophobic target-binding patches

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Increased levels of ligands of Toll-like receptors 2 and 4 in type 1 diabetes

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Prevention of diabetic nephropathy in db/db mice with glycated albumin antagonists. A novel treatment strategy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Nonenzymatic Glucosylation of Serum Proteins in Diabetes Mellitus

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Mass spectrometric monitoring of albumin in uremia

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

High mobility group box 1 protein interacts with multiple Toll-like receptors

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Serum carboxymethyllysine concentrations are reduced in diabetic men with abdominal aortic aneurysms: Health In Men Study.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.134

7 January 2021

Identifiers

DOI

10.1038/KI.2010.134

1 reference

17 October 2017

1 reference