Endoplasmic reticulum D-myo-inositol 1,4,5-trisphosphate-sensitive stores regulate nuclear factor-kappaB binding activity in a calcium-independent manner (Q42502990)

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English	Endoplasmic reticulum D-myo-inositol 1,4,5-trisphosphate-sensitive stores regulate nuclear factor-kappaB binding activity in a calcium-independent manner	scientific article

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scholarly article

1 reference

Europe PubMed Central

1 November 2017

Endoplasmic reticulum D-myo-inositol 1,4,5-trisphosphate-sensitive stores regulate nuclear factor-kappaB binding activity in a calcium-independent manner (English)

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Europe PubMed Central

1 November 2017

endoplasmic reticulum

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4

object named as

Mattson MP

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Europe PubMed Central

1 November 2017

author name string

Glazner GW

1

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Europe PubMed Central

1 November 2017

Camandola S

2

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Europe PubMed Central

1 November 2017

Geiger JD

3

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Europe PubMed Central

1 November 2017

language of work or name

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publication date

17 April 2001

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Europe PubMed Central

1 November 2017

Journal of Biological Chemistry

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Europe PubMed Central

1 November 2017

276

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Europe PubMed Central

1 November 2017

25

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Europe PubMed Central

1 November 2017

22461-22467

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Europe PubMed Central

1 November 2017

NF-kappa B: ten years after

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Constitutive NF-kappa B activity in neurons

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Nuclear factor-kappa B in rat brain: enhanced DNA-binding activity following convulsant-induced seizures.

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Global cerebral ischemia activates nuclear factor-kappa B prior to evidence of DNA fragmentation.

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NF-kappaB is activated and promotes cell death in focal cerebral ischemia.

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Transcription factor NF-kappaB is activated in primary neurons by amyloid beta peptides and in neurons surrounding early plaques from patients with Alzheimer disease

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Redox signals and NF-kappaB activation in T cells.

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Intracellular oxidation/reduction status in the regulation of transcription factors NF-kappaB and AP-1.

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NF-kappaB as a primary regulator of the stress response

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Regulation of neuronal function by calcium

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Neuronal calcium signaling

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Calcium-induced calcium release in neurones

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The ER-overload response: activation of NF-kappa B.

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Activation of DNA-binding activity in an apparently cytoplasmic precursor of the NF-kappa B transcription factor

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Xestospongins: potent membrane permeable blockers of the inositol 1,4,5-trisphosphate receptor

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Requirement of the inositol trisphosphate receptor for activation of store-operated Ca2+ channels

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Thapsigargin, a tumor promoter, discharges intracellular Ca2+ stores by specific inhibition of the endoplasmic reticulum Ca2(+)-ATPase

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Kinetics of thapsigargin-Ca(2+)-ATPase (sarcoplasmic reticulum) interaction reveals a two-step binding mechanism and picomolar inhibition

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CREB and memory consolidation

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The matter of mind: molecular control of memory

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Regulation of gene expression in hippocampal neurons by distinct calcium signaling pathways

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Brain synapses contain inducible forms of the transcription factor NF-kappa B.

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Nuclear factor-kappaB mediates the cell survival-promoting action of activity-dependent neurotrophic factor peptide-9

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Increased activation of nuclear factor kappa B in rat lumbar dorsal root ganglion neurons following partial sciatic nerve injuries.

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NF-kappa B: a crucial transcription factor for glial and neuronal cell function

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Gene expression of the transcription factor NF-kappa B in hippocampus: regulation by synaptic activity

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Involvement of intracellular Ca2+ in oxidant-induced NF-kappa B activation

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Spontaneous calcium release from inositol trisphosphate-sensitive calcium stores.

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Calcium from internal stores modifies dendritic spine shape

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Roles of thapsigargin-sensitive Ca2+ stores in the survival of developing cultured neurons.

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p75-mediated NF-kappaB activation enhances the survival response of developing sensory neurons to nerve growth factor.

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Glutamate-stimulated production of inositol phosphates is mediated by Ca2+ influx in oligodendrocyte progenitors

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10.1074/JBC.M101315200

1 reference

Europe PubMed Central

1 November 2017

1 reference

Europe PubMed Central

1 November 2017

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