Functional inhibition of PI3K by the betaGBP molecule suppresses Ras-MAPK signalling to block cell proliferation (Q42515047)

1 November 2017

Functional inhibition of PI3K by the betaGBP molecule suppresses Ras-MAPK signalling to block cell proliferation (English)

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1 November 2017

2

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1 November 2017

Wells V

1

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1 November 2017

Mallucci L

series ordinal

3

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1 November 2017

language of work or name

English

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publication date

2 July 2007

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1 November 2017

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1 November 2017

26

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55

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1 November 2017

7709-7714

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1210580

1 November 2017

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cites work

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The phosphatidylinositol 3-Kinase AKT pathway in human cancer

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Role of phosphoinositide 3-kinase in activation of ras and mitogen-activated protein kinase by epidermal growth factor

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Translocation involving chromosome 22 in Ewing's sarcoma. A cytogenetic study of four fresh tumors

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Mapping on Human and Mouse Chromosomes of the Gene for the β-Galactoside-Binding Protein, an Autocrine-Negative Growth Factor

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Beta-galactoside-binding protein secreted by activated T cells inhibits antigen-induced proliferation of T cells

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Chromosomal abnormalities in clear cell sarcoma. Implications for histogenesis

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The phosphoinositide 3-kinase pathway

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Targeting RAS signalling pathways in cancer therapy

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Association of pRas and pRaf-1 in a complex correlates with activation of a signal transduction pathway

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Phosphoinositide 3-kinase controls early and late events in mammalian cell division

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Phosphatidylinositol 3-kinase is an early intermediate in the G beta gamma-mediated mitogen-activated protein kinase signaling pathway

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Exploiting the PI3K/AKT pathway for cancer drug discovery

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Production and purification of a recombinant human 14 kDa beta-galactoside-binding lectin

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A family of proteins that inhibit signalling through tyrosine kinase receptors

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Direct evidence that Gi-coupled receptor stimulation of mitogen-activated protein kinase is mediated by G beta gamma activation of p21ras

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Gbetagamma stimulates phosphoinositide 3-kinase-gamma by direct interaction with two domains of the catalytic p110 subunit.

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Turning cell cycle controller genes into cancer drugs. A role for an antiproliferative cytokine (betaGBP).

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Signaling interplay in Ras superfamily function

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Circumventing multidrug resistance in cancer by beta-galactoside binding protein, an antiproliferative cytokine

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Abnormalities of chromosome 22 in human brain tumors determined by combined cytogenetic and molecular genetic approaches.

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Phosphatidylinositol-3-OH kinase as a direct target of Ras

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p84, a new Gbetagamma-activated regulatory subunit of the type IB phosphoinositide 3-kinase p110gamma

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Recurrent breakpoints at 9q31 and 22q12.2 in extraskeletal myxoid chondrosarcoma

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Synthesis and function of 3-phosphorylated inositol lipids

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7 January 2021

Phosphoinositide 3-kinases: a conserved family of signal transducers

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7 January 2021

Structural Determinants of Phosphoinositide 3-Kinase Inhibition by Wortmannin, LY294002, Quercetin, Myricetin, and Staurosporine

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7 January 2021

Identification of an autocrine negative growth factor: mouse beta-galactoside-binding protein is a cytostatic factor and cell growth regulator

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Molecular expression of the negative growth factor murine beta-galactoside binding protein (mGBP)

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1210580

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Dimensions Publication ID

1 November 2017

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