Copper modulation of ion channels of PrP[106-126] mutant prion peptide fragments (Q42604287)

3 November 2017

title

Copper modulation of ion channels of PrP[106-126] mutant prion peptide fragments (English)

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3 November 2017

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8

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3 November 2017

J I Kourie

1

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3 November 2017

B L Kenna

2

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3 November 2017

D Tew

3

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3 November 2017

M F Jobling

4

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3 November 2017

C C Curtain

5

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3 November 2017

C L Masters

6

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3 November 2017

K J Barnham

7

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3 November 2017

publication date

1 May 2003

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3 November 2017

number of pages

11

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Journal of Membrane Biology

inferred from page(s)

published in

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3 November 2017

volume

193

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3 November 2017

issue

1

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3 November 2017

page(s)

35-45

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Channel formation by a neurotoxic prion protein fragment

3 November 2017

cites work

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12 December 2020

JPCalc, a software package for calculating liquid junction potential corrections in patch-clamp, intracellular, epithelial and bilayer measurements and for correcting junction potential measurements

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12 December 2020

Involvement of the 5-lipoxygenase pathway in the neurotoxicity of the prion peptide PrP106-126.

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12 December 2020

Ca++-induced fusion of fragmented sarcoplasmic reticulum with artificial planar bilayers

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12 December 2020

Theoretical models of the ion channel structure of amyloid beta-protein

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12 December 2020

Mechanisms of prion-induced modifications in membrane transport properties: implications for signal transduction and neurotoxicity

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12 December 2020

Intrinsic physiological and morphological properties of principal cells of the hippocampus and neocortex in hamsters infected with scrapie

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12 December 2020

Antioxidant activity related to copper binding of native prion protein

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12 December 2020

Channel activity of deamidated isoforms of prion protein fragment 106-126 in planar lipid bilayers

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12 December 2020

Calcium dependence of C-type natriuretic peptide-formed fast K(+) channel

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12 December 2020

Zn2+ interaction with Alzheimer amyloid beta protein calcium channels

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12 December 2020

Structure-activity analyses of beta-amyloid peptides: contributions of the beta 25-35 region to aggregation and neurotoxicity

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12 December 2020

Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

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12 December 2020

The conformational preference of gramicidin channels is a function of lipid bilayer thickness

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12 December 2020

Neurodegenerative disease: amyloid pores from pathogenic mutations

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12 December 2020

Metals and neuroscience.

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12 December 2020

Solution Structures of β Peptide and Its Constituent Fragments: Relation to Amyloid Deposition

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12 December 2020

Conversion of alpha-helices into beta-sheets features in the formation of the scrapie prion proteins

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12 December 2020

Ion channel formation and membrane-linked pathologies of misfolded hydrophobic proteins: the role of dangerous unchaperoned molecules

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12 December 2020

Prion peptide fragment PrP[106-126] forms distinct cation channel types.

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12 December 2020

Copper and zinc binding modulates the aggregation and neurotoxic properties of the prion peptide PrP106-126.

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12 December 2020

Nerve growth factor does not influence the expression of beta amyloid precursor protein mRNA in rat brain: in vivo and in vitro studies

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12 December 2020

Intracellular calcium rise through L-type calcium channels, as molecular mechanism for prion protein fragment 106-126-induced astroglial proliferation.

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12 December 2020

Protein aggregation and deposition: implications for ion channel formation and membrane damage.

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12 December 2020

Prion disease: A loss of antioxidant function?

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12 December 2020

Prion peptide 106-126 as a model for prion replication and neurotoxicity

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12 December 2020

Solution conformations and aggregational properties of synthetic amyloid beta-peptides of Alzheimer's disease. Analysis of circular dichroism spectra

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12 December 2020

Characteristics of two types of chloride channel in sarcoplasmic reticulum vesicles from rabbit skeletal muscle

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12 December 2020

Substitutions of hydrophobic amino acids reduce the amyloidogenicity of Alzheimer's disease beta A4 peptides

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12 December 2020

Modelling charge interactions in the prion protein: predictions for pathogenesis

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12 December 2020

Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neurons.

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12 December 2020

Measuring kinetics of complex single ion channel data using mean-variance histograms

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12 December 2020

The hydrophobic core sequence modulates the neurotoxic and secondary structure properties of the prion peptide 106-126.

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12 December 2020

Prion peptide 106-126 modulates the aggregation of cellular prion protein and induces the synthesis of potentially neurotoxic transmembrane PrP

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12 December 2020

Amyloid peptide channels: blockade by zinc and inhibition by Congo red (amyloid channel block).

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12 December 2020

Theoretical analysis of the implication of PrP in neuronal death during transmissible subacute spongiform encephalopathies: hypothesis of a PrP oligomeric channel.

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12 December 2020

Alzheimer's beta-amyloid, human islet amylin, and prion protein fragment evoke intracellular free calcium elevations by a common mechanism in a hypothalamic GnRH neuronal cell line

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12 December 2020

Studies on peptide fragments of prion proteins.

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12 December 2020

Mouse cortical cells lacking cellular PrP survive in culture with a neurotoxic PrP fragment

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12 December 2020

Diversity of amyloid beta protein fragment [1-40]-formed channels.

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12 December 2020

Prion Protein Biology

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12 December 2020

10.1007/S00232-002-2005-5

Identifiers

DOI

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3 November 2017

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