Cyclic AMP inhibits JNK activation by CREB-mediated induction of c-FLIP(L) and MKP-1, thereby antagonizing UV-induced apoptosis (Q42811539)

7 November 2017

Cyclic AMP inhibits JNK activation by CREB-mediated induction of c-FLIP(L) and MKP-1, thereby antagonizing UV-induced apoptosis (English)

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7 November 2017

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J Zhang

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Q Wang

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N Zhu

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M Yu

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B Shen

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J Xiang

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A Lin

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20 June 2008

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Cell Death & Differentiation

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1654-1662

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https://scigraph.springernature.com/pub.10.1038/cdd.2008.87

7 November 2017

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JNK suppresses apoptosis via phosphorylation of the proapoptotic Bcl-2 family protein BAD

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Epac is a Rap1 guanine-nucleotide-exchange factor directly activated by cyclic AMP

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cAMP-PKA signaling to the mitochondria: protein scaffolds, mRNA and phosphatases

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Transcriptional regulation by the phosphorylation-dependent factor CREB

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Defining the CREB regulon: a genome-wide analysis of transcription factor regulatory regions.

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Genome-wide analysis of cAMP-response element binding protein occupancy, phosphorylation, and target gene activation in human tissues

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TNF-alpha is the critical mediator of the cyclic AMP-induced apoptosis of CD8+4+ double-positive thymocytes.

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Neutrophil apoptosis mediated by nicotinic acid receptors (GPR109A)

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Cyclic AMP inhibits p38 activation via CREB-induced dynein light chain.

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Crosstalk between cAMP and MAP kinase signaling in the regulation of cell proliferation

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Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

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A peptide inhibitor of c-Jun N-terminal kinase protects against excitotoxicity and cerebral ischemia

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CAMP-dependent protein kinase enhances CYP17 transcription via MKP-1 activation in H295R human adrenocortical cells

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Bile Acid Regulation of C/EBPβ, CREB, and c-Jun Function, via the Extracellular Signal-Regulated Kinase and c-Jun NH2-Terminal Kinase Pathways, Modulates the Apoptotic Response of Hepatocytes

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NF-kappaB at the crossroads of life and death

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Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

21 January 2018

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Macrophage glucocorticoid receptors regulate Toll-like receptor 4-mediated inflammatory responses by selective inhibition of p38 MAP kinase

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Conditional expression of mitogen-activated protein kinase phosphatase-1, MKP-1, is cytoprotective against UV-induced apoptosis

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Inhibition of death receptor signals by cellular FLIP

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An antiapoptotic protein, c-FLIPL, directly binds to MKK7 and inhibits the JNK pathway

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Activation of cyclic AMP pathway prevents CD34(+) cell apoptosis

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7 January 2021

c-Jun N-terminal kinase but not mitogen-activated protein kinase is sensitive to cAMP inhibition in T lymphocytes

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7 January 2021

Cyclic AMP inhibition of thrombin-induced growth in vascular smooth muscle cells correlates with decreased JNK1 activity and c-Jun expression

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7 January 2021

Cyclic AMP-elevating agents induce the expression of MAP kinase phosphatase-1 in PC12 cells

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https://api.crossref.org/works/10.1038%2FCDD.2008.87

7 January 2021

NF-kappaB is required for UV-induced JNK activation via induction of PKCdelta

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https://api.crossref.org/works/10.1038%2FCDD.2008.87

7 January 2021

Mitogen-activated protein kinase phosphatase-1 (MKP-1) expression is induced by low oxygen conditions found in solid tumor microenvironments. A candidate MKP for the inactivation of hypoxia-inducible stress-activated protein kinase/c-Jun N-terminal

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2008.87

7 January 2021

Identifiers

DOI

10.1038/CDD.2008.87

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Dimensions Publication ID

7 November 2017

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