Tumor necrosis factor alpha (TNFalpha) stimulates Map4k4 expression through TNFalpha receptor 1 signaling to c-Jun and activating transcription factor 2. (Q42828385)

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scientific article published on 11 May 2007

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English	Tumor necrosis factor alpha (TNFalpha) stimulates Map4k4 expression through TNFalpha receptor 1 signaling to c-Jun and activating transcription factor 2.	scientific article published on 11 May 2007

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scholarly article

1 reference

Europe PubMed Central

8 November 2017

Tumor necrosis factor alpha (TNFalpha) stimulates Map4k4 expression through TNFalpha receptor 1 signaling to c-Jun and activating transcription factor 2. (English)

1 reference

Europe PubMed Central

8 November 2017

author name string

Gregory J Tesz

1

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Europe PubMed Central

8 November 2017

Adilson Guilherme

2

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Europe PubMed Central

8 November 2017

Kalyani V P Guntur

3

1 reference

Europe PubMed Central

8 November 2017

Andrea C Hubbard

4

1 reference

Europe PubMed Central

8 November 2017

Xiaoqing Tang

5

1 reference

Europe PubMed Central

8 November 2017

Anil Chawla

6

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Europe PubMed Central

8 November 2017

Michael P Czech

7

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Europe PubMed Central

8 November 2017

language of work or name

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publication date

11 May 2007

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Europe PubMed Central

8 November 2017

Journal of Biological Chemistry

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Europe PubMed Central

8 November 2017

282

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Europe PubMed Central

8 November 2017

27

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Europe PubMed Central

8 November 2017

19302-19312

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Europe PubMed Central

8 November 2017

Mitogen-activated protein kinase signaling

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M700665200

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C-Jun N-terminal kinases/c-Jun and p38 pathways cooperate in ceramide-induced neuronal apoptosis.

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p38 mitogen-activated protein kinase activity commits embryonic stem cells to either neurogenesis or cardiomyogenesis

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Inhibition of p38MAPK increases adipogenesis from embryonic to adult stages

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The role of MAPKs in adipocyte differentiation and obesity.

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ERK and p38 MAPK-activated protein kinases: a family of protein kinases with diverse biological functions

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An RNA interference-based screen identifies MAP4K4/NIK as a negative regulator of PPARgamma, adipogenesis, and insulin-responsive hexose transport

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The Traf2- and Nck-interacting kinase as a putative effector of Rap2 to regulate actin cytoskeleton

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Identification and functional characterization of a novel human misshapen/Nck interacting kinase-related kinase, hMINK beta

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p38 and a p38-interacting protein are critical for downregulation of E-cadherin during mouse gastrulation

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A small interfering RNA screen for modulators of tumor cell motility identifies MAP4K4 as a promigratory kinase

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The STE20 kinase HGK is broadly expressed in human tumor cells and can modulate cellular transformation, invasion, and adhesion

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MAP4K4 gene silencing in human skeletal muscle prevents tumor necrosis factor-alpha-induced insulin resistance.

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Transcriptional control of adipocyte formation

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Adipose-selective targeting of the GLUT4 gene impairs insulin action in muscle and liver

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Targeted disruption of the glucose transporter 4 selectively in muscle causes insulin resistance and glucose intolerance.

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Tumor necrosis factor-alpha suppresses adipocyte-specific genes and activates expression of preadipocyte genes in 3T3-L1 adipocytes: nuclear factor-kappaB activation by TNF-alpha is obligatory

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Profiling gene transcription in vivo reveals adipose tissue as an immediate target of tumor necrosis factor-alpha: implications for insulin resistance

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A novel human STE20-related protein kinase, HGK, that specifically activates the c-Jun N-terminal kinase signaling pathway

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21 January 2018

Inhibition of insulin receptor signaling by TNF: potential role in obesity and non-insulin-dependent diabetes mellitus

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Reduced tyrosine kinase activity of the insulin receptor in obesity-diabetes. Central role of tumor necrosis factor-alpha

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Tumor necrosis factor alpha inhibits signaling from the insulin receptor

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IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistance

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Interleukin-1beta-induced insulin resistance in adipocytes through down-regulation of insulin receptor substrate-1 expression

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Altered gene expression for tumor necrosis factor-alpha and its receptors during drug and dietary modulation of insulin resistance.

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Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance

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Increased adipose tissue expression of tumor necrosis factor-alpha in human obesity and insulin resistance

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The expression of TNF alpha by human muscle. Relationship to insulin resistance

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Tumor necrosis factor-alpha induces skeletal muscle insulin resistance in healthy human subjects via inhibition of Akt substrate 160 phosphorylation

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Tumor necrosis factor (TNF)-alpha inhibits insulin signaling through stimulation of the p55 TNF receptor and activation of sphingomyelinase

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TNF-alpha induction of lipolysis is mediated through activation of the extracellular signal related kinase pathway in 3T3-L1 adipocytes

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Insulin signaling through Akt/protein kinase B analyzed by small interfering RNA-mediated gene silencing

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Glucose transporter recycling in response to insulin is facilitated by myosin Myo1c

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A PCR primer bank for quantitative gene expression analysis

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Transcription factor ATF2 regulation by the JNK signal transduction pathway

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Signaling pathways utilized by tumor necrosis factor receptor 1 in adipocytes to suppress differentiation

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Identifiers

10.1074/JBC.M700665200

1 reference

Europe PubMed Central

8 November 2017

1 reference

Europe PubMed Central

8 November 2017

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