Neuropeptide Y suppresses anorexigenic output from the ventromedial nucleus of the hypothalamus. (Q43142660)

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19 January 2020

title

Neuropeptide Y suppresses anorexigenic output from the ventromedial nucleus of the hypothalamus (English)

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20203197%20AND%20SRC:MED&resulttype=core&format=json

19 January 2020

author name string

Melissa J S Chee

1

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19 January 2020

Martin G Myers

2

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19 January 2020

Christopher J Price

3

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19 January 2020

William F Colmers

4

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19 January 2020

language of work or name

English

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publication date

1 March 2010

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19 January 2020

published in

Journal of Neuroscience

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19 January 2020

volume

30

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19 January 2020

issue

9

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19 January 2020

page(s)

3380-3390

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Characterization of the neuroanatomical distribution of agouti-related protein immunoreactivity in the rhesus monkey and the rat.

19 January 2020

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Leptin activates anorexigenic POMC neurons through a neural network in the arcuate nucleus

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Convergence of pre- and postsynaptic influences on glucosensing neurons in the ventromedial hypothalamic nucleus

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Physiological and molecular characteristics of rat hypothalamic ventromedial nucleus glucosensing neurons

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Neuropeptide Y-mediated inhibition of proopiomelanocortin neurons in the arcuate nucleus shows enhanced desensitization in ob/ob mice.

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Neuropeptide Y inhibits hypocretin/orexin neurons by multiple presynaptic and postsynaptic mechanisms: tonic depression of the hypothalamic arousal system.

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Spontaneous release of neuropeptide Y tonically inhibits recurrent mossy fiber synaptic transmission in epileptic brain

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Mechanisms of neuropeptide Y, peptide YY, and pancreatic polypeptide inhibition of identified green fluorescent protein-expressing GABA neurons in the hypothalamic neuroendocrine arcuate nucleus.

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The anti-epileptic actions of neuropeptide Y in the hippocampus are mediated by Y and not Y receptors

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The rise, fall, and resurrection of the ventromedial hypothalamus in the regulation of feeding behavior and body weight

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Leptin directly activates SF1 neurons in the VMH, and this action by leptin is required for normal body-weight homeostasis

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Substantia Gelatinosa neurons in defined-medium organotypic slice culture are similar to those in acute slices from young adult rats

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Structure-function analysis of stimulation of food intake by neuropeptide Y: Effects of receptor agonists

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Sciatic chronic constriction injury produces cell-type-specific changes in the electrophysiological properties of rat substantia gelatinosa neurons.

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10.1523/JNEUROSCI.4031-09.2010

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DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20203197%20AND%20SRC:MED&resulttype=core&format=json

19 January 2020

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19 January 2020

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