Histamine acting on H1 receptor promotes inhibition of proliferation via PLC, RAC, and JNK-dependent pathways (Q43242549)

17 November 2017

Histamine acting on H1 receptor promotes inhibition of proliferation via PLC, RAC, and JNK-dependent pathways (English)

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17 November 2017

Cintia Notcovich

1

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17 November 2017

Federico Diez

2

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17 November 2017

Maria Rosario Tubio

3

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17 November 2017

Alberto Baldi

4

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17 November 2017

Marcelo G Kazanietz

5

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17 November 2017

Carlos Davio

6

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17 November 2017

Carina Shayo

7

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17 November 2017

language of work or name

English

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publication date

11 November 2009

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Experimental Cell Research

17 November 2017

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17 November 2017

316

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17 November 2017

401-411

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17 November 2017

3

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Model systems for the study of seven-transmembrane-segment receptors

17 November 2017

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Potent activation of RhoA by Galpha q and Gq-coupled receptors.

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Small GTP-binding protein-coupled receptors

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Mepyramine, a histamine H1 receptor inverse agonist, binds preferentially to a G protein-coupled form of the receptor and sequesters G protein.

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Histamine induces tissue factor expression: implications for acute coronary syndromes

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Epidermal growth factor differentially augments G(i)-mediated stimulation of c-Jun N-terminal kinase activity

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Conditionally expressed G alpha 15 couples to endogenous receptors in GH3 cells.

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A Rac/Cdc42-specific exchange factor, GEFT, induces cell proliferation, transformation, and migration

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The pleckstrin homology domain of phospholipase C-beta2 as an effector site for Rac.

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Stimulation of Galphaq-coupled M1 muscarinic receptor causes reversible spectrin redistribution mediated by PLC, PKC and ROCK.

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Rho-regulated signals induce apoptosis in vitro and in vivo by a p53-independent, but Bcl2 dependent pathway.

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Activation of the p38 and p42/p44 mitogen-activated protein kinase families by the histamine H(1) receptor in DDT(1)MF-2 cells

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10.1016/J.YEXCR.2009.11.002

7 January 2021

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DOI

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17 November 2017

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