IL-4Rα-responsive smooth muscle cells contribute to initiation of TH2 immunity and pulmonary pathology in Nippostrongylus brasiliensis infections. (Q43592011)

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scientific article published on 25 August 2010

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English	IL-4Rα-responsive smooth muscle cells contribute to initiation of TH2 immunity and pulmonary pathology in Nippostrongylus brasiliensis infections.	scientific article published on 25 August 2010

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11 February 2020

IL-4Rα-responsive smooth muscle cells contribute to initiation of TH2 immunity and pulmonary pathology in Nippostrongylus brasiliensis infections (English)

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11 February 2020

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Nippostrongylus brasiliensis

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Nippostrongylus brasiliensis

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11 February 2020

Antony J Cutler

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11 February 2020

Benjamin G Dewals

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11 February 2020

Elmarie Myburgh

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11 February 2020

Andreas L Lopata

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11 February 2020

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W G C Horsnell

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11 February 2020

A Vira

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11 February 2020

F Kirstein

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11 February 2020

J C Hoving

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11 February 2020

M Kimberg

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11 February 2020

B Arendse

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11 February 2020

N White

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11 February 2020

P E Burger

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11 February 2020

F Brombacher

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11 February 2020

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25 August 2010

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11 February 2020

Mucosal Immunology

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11 February 2020

4

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11 February 2020

1

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11 February 2020

83-92

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11 February 2020

https://scigraph.springernature.com/pub.10.1038/mi.2010.46

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Essential role of Stat6 in IL-4 signalling

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IL-13, IL-4Ralpha, and Stat6 are required for the expulsion of the gastrointestinal nematode parasite Nippostrongylus brasiliensis.

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IL-4 receptor signaling in Clara cells is required for allergen-induced mucus production

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IL-4R{alpha}-responsive smooth muscle cells increase intestinal hypercontractility and contribute to resistance during acute Schistosomiasis.

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Anti-inflammatory responses and oxidative stress in Nippostrongylus brasiliensis-induced pulmonary inflammation

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Nippostrongylus brasiliensis infection leads to the development of emphysema associated with the induction of alternatively activated macrophages

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Eosinophil deficiency compromises parasite survival in chronic nematode infection.

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Interleukin-4-promoted T helper 2 responses enhance Nippostrongylus brasiliensis-induced pulmonary pathology.

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