Modulation of double-stranded RNA-activated protein kinase in insulin sensitive tissues of obese humans. (Q43699807)

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26 February 2020

title

Modulation of double-stranded RNA-activated protein kinase in insulin sensitive tissues of obese humans (English)

1 reference

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26 February 2020

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4

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26 February 2020

Bruno M Carvalho

1

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26 February 2020

Alexandre G Oliveira

2

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26 February 2020

José B Carvalheira

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26 February 2020

author name string

Mirian Ueno

3

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26 February 2020

Dioze Guadagnini

5

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26 February 2020

Marco A Carvalho-Filho

6

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26 February 2020

Bruno Geloneze

7

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26 February 2020

Marcelo M O Lima

8

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26 February 2020

Jose C Pareja

9

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26 February 2020

Mario J A Saad

11

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26 February 2020

publication date

11 June 2013

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26 February 2020

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26 February 2020

volume

21

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26 February 2020

issue

12

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26 February 2020

page(s)

2452-2457

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The cellular and signaling networks linking the immune system and metabolism in disease

26 February 2020

cites work

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Elevated toll-like receptor 4 expression and signaling in muscle from insulin-resistant subjects

21 January 2018

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Endoplasmic Reticulum Stress and Inflammation in Obesity and Diabetes

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Double-stranded RNA-dependent protein kinase links pathogen sensing with stress and metabolic homeostasis

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Double-stranded RNA-activated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice.

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Western diet modulates insulin signaling, c-Jun N-terminal kinase activity, and insulin receptor substrate-1ser307 phosphorylation in a tissue-specific fashion

21 January 2018

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21 January 2018

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The unfolded protein response in human skeletal muscle is not involved in the onset of glucose tolerance impairment induced by a fat-rich diet.

21 January 2018

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Impaired regulation of the TNF-alpha converting enzyme/tissue inhibitor of metalloproteinase 3 proteolytic system in skeletal muscle of obese type 2 diabetic patients: a new mechanism of insulin resistance in humans

21 January 2018

1 reference

Poly(I-C)-induced Toll-like receptor 3 (TLR3)-mediated activation of NFkappa B and MAP kinase is through an interleukin-1 receptor-associated kinase (IRAK)-independent pathway employing the signaling components TLR3-TRAF6-TAK1-TAB2-PKR

21 January 2018

1 reference