Neuroprotective effects of the N-terminal tripeptide of IGF-1, glycine-proline-glutamate, in the immature rat brain after hypoxic-ischemic injury (Q43813696)

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scientific article published in December 2001

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English	Neuroprotective effects of the N-terminal tripeptide of IGF-1, glycine-proline-glutamate, in the immature rat brain after hypoxic-ischemic injury	scientific article published in December 2001

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

27 November 2017

Neuroprotective effects of the N-terminal tripeptide of IGF-1, glycine-proline-glutamate, in the immature rat brain after hypoxic-ischemic injury (English)

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Europe PubMed Central

PubMed publication ID

27 November 2017

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4

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Gluckman PD

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Europe PubMed Central

PubMed publication ID

27 November 2017

author name string

Sizonenko SV

1

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Europe PubMed Central

PubMed publication ID

27 November 2017

Sirimanne ES

2

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Europe PubMed Central

PubMed publication ID

27 November 2017

Williams CE

3

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Europe PubMed Central

PubMed publication ID

27 November 2017

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publication date

1 December 2001

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Europe PubMed Central

PubMed publication ID

27 November 2017

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Europe PubMed Central

PubMed publication ID

27 November 2017

922

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Europe PubMed Central

PubMed publication ID

27 November 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

27 November 2017

42-50

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Europe PubMed Central

PubMed publication ID

27 November 2017

Peripheral infusion of IGF-I selectively induces neurogenesis in the adult rat hippocampus.

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Insulin-like growth factor II is induced during wound repair following hypoxic-ischemic injury in the developing rat brain

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Co-ordinated and cellular specific induction of the components of the IGF/IGFBP axis in the rat brain following hypoxic-ischemic injury

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Role of insulin-like growth factor binding proteins in limitation of IGF-I degradation into the N-methyl-D-aspartate receptor antagonist GPE: evidence from gonadotrophin-releasing hormone secretion in vitro at two developmental stages.

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Immediate-early gene protein expression in neurons undergoing delayed death, but not necrosis, following hypoxic-ischaemic injury to the young rat brain

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Apoptosis in cerebellar granule cells is blocked by high KCl, forskolin, and IGF-1 through distinct mechanisms of action: the involvement of intracellular calcium and RNA synthesis

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Intracerebral transportation and cellular localisation of insulin-like growth factor-1 following central administration to rats with hypoxic-ischemic brain injury.

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The effects of insulin-like growth factor (IGF)-1, IGF-2, and des-IGF-1 on neuronal loss after hypoxic-ischemic brain injury in adult rats: evidence for a role for IGF binding proteins

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10.1016/S0006-8993(01)03148-1

1 reference

Europe PubMed Central

PubMed publication ID

27 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

27 November 2017

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