Lactate administration attenuates cognitive deficits following traumatic brain injury (Q43885977)

27 November 2017

title

Lactate administration attenuates cognitive deficits following traumatic brain injury (English)

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27 November 2017

main subject

traumatic brain injury

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author name string

Ann C Rice

1

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27 November 2017

Robert Zsoldos

2

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27 November 2017

Tao Chen

3

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27 November 2017

Margaret S Wilson

4

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27 November 2017

B Alessandri

5

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27 November 2017

Robert J Hamm

6

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27 November 2017

M Ross Bullock

7

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27 November 2017

language of work or name

English

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publication date

1 February 2002

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27 November 2017

number of pages

4

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based on heuristic

inferred from page(s)

published in

Brain Research

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27 November 2017

volume

928

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27 November 2017

issue

1-2

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27 November 2017

page(s)

156-159

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Functional coupling between neurons and glia.

27 November 2017

cites work

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Spatial learning impairment parallels the magnitude of dorsal hippocampal lesions, but is hardly present following ventral lesions.

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Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization

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Brain lactate, not glucose, fuels the recovery of synaptic function from hypoxia upon reoxygenation: an in vitro study

7 January 2021

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Brain anaerobic lactate production: a suicide note or a survival kit?

7 January 2021

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Evaluation of Memory Dysfunction Following Experimental Brain Injury Using the Morris Water Maze

7 January 2021

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Partitioning of CO2 production between glucose and lactate in excised sympathetic ganglia, with implications for brain

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Identifiers

DOI

10.1016/S0006-8993(01)03299-1

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27 November 2017

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