Chronic application of MTII in a rat model of obesity results in sustained weight loss (Q43911366)

28 November 2017

title

Chronic application of MTII in a rat model of obesity results in sustained weight loss (English)

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28 November 2017

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Bradford S Hamilton

1

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28 November 2017

Henri N Doods

series ordinal

2

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28 November 2017

publication date

1 March 2002

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28 November 2017

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28 November 2017

volume

10

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28 November 2017

issue

3

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28 November 2017

page(s)

182-187

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Treatment of obesity by moderate and severe caloric restriction. Results of clinical research trials

28 November 2017

cites work

1 reference

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Ectopic expression of the agouti gene in transgenic mice causes obesity, features of type II diabetes, and yellow fur

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Targeted disruption of the melanocortin-4 receptor results in obesity in mice

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Hyperproinsulinaemia in obese fat/fat mice associated with a carboxypeptidase E mutation which reduces enzyme activity

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A C-terminal fragment of Agouti-related protein increases feeding and antagonizes the effect of alpha-melanocyte stimulating hormone in vivo

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Altered energy balance causes selective changes in melanocortin-4(MC4-R), but not melanocortin-3 (MC3-R), receptors in specific hypothalamic regions: further evidence that activation of MC4-R is a physiological inhibitor of feeding

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31 March 2020

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31 March 2020

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Melanocortin mediated inhibition of feeding behavior in rats

31 March 2020

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Centrally administered MTII affects feeding, drinking, temperature, and activity in the Sprague-Dawley rat.

31 March 2020

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Selective antagonist for the melanocortin 4 receptor (HS014) increases food intake in free-feeding rats

31 March 2020

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Role of the melanocortin-4 receptor in metabolic rate and food intake in mice

31 March 2020

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31 March 2020

Identifiers

DOI

10.1038/OBY.2002.28

1 reference

28 November 2017

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