Different mechanisms are used by insulin to repress three genes that contain a homologous thymine-rich insulin response element (Q44546920)

4 December 2017

title

Different mechanisms are used by insulin to repress three genes that contain a homologous thymine-rich insulin response element (English)

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4 December 2017

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3

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4 December 2017

Satish Patel

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4 December 2017

Christopher Lipina

series ordinal

2

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4 December 2017

language of work or name

English

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publication date

1 August 2003

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4 December 2017

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4 December 2017

volume

549

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4 December 2017

issue

1-3

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4 December 2017

page(s)

72-76

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Differential expression of insulin-like growth factor-I and insulin-like growth factor binding protein-1 in the diabetic rat

4 December 2017

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Insulin regulation of phosphoenolpyruvate carboxykinase gene expression does not require activation of the Ras/mitogen-activated protein kinase signaling pathway

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Central role for phosphatidylinositide 3-kinase in the repression of glucose-6-phosphatase gene transcription by insulin

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Phosphatidylinositol 3'-kinase and p70s6k are required for insulin but not bisperoxovanadium 1,10-phenanthroline (bpV(phen)) inhibition of insulin-like growth factor binding protein gene expression. Evidence for MEK-independent activation of mitogen

21 January 2018

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Insulin regulation of insulin-like growth factor-binding protein-1 gene expression is dependent on the mammalian target of rapamycin, but independent of ribosomal S6 kinase activity

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Tor proteins and protein phosphatase 2A reciprocally regulate Tap42 in controlling cell growth in yeast

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TOR, a central controller of cell growth

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Antagonistic effects of phorbol esters on insulin regulation of insulin-like growth factor-binding protein-1 (IGFBP-1) but not glucose-6-phosphatase gene expression

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TIP41 interacts with TAP42 and negatively regulates the TOR signaling pathway

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Expression and chromosomal localization of the human alpha 4/IGBP1 gene, the structure of which is closely related to the yeast TAP42 protein of the rapamycin-sensitive signal transduction pathway

21 January 2018

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Amino acid sufficiency and mTOR regulate p70 S6 kinase and eIF-4E BP1 through a common effector mechanism

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Protein phosphatase 2A interacts with the 70-kDa S6 kinase and is activated by inhibition of FKBP12-rapamycinassociated protein

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Mammalian TOR controls one of two kinase pathways acting upon nPKCdelta and nPKCepsilon

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Inactivation of glycogen synthase kinase-3 by epidermal growth factor is mediated by mitogen-activated protein kinase/p90 ribosomal protein S6 kinase signaling pathway in NIH/3T3 cells

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Oxidative and chemical stress mimic insulin by selectively inhibiting the expression of phosphoenolpyruvate carboxykinase in hepatoma cells

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Global and societal implications of the diabetes epidemic

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New drug targets for type 2 diabetes and the metabolic syndrome

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Phosphatidylinositol 3-Kinase, but Not p70/p85 Ribosomal S6 Protein Kinase, Is Required for the Regulation of Phosphoenolpyruvate Carboxykinase (PEPCK) Gene Expression by Insulin

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7 January 2021

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7 January 2021

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7 January 2021

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5-aminoimidazole-4-carboxamide riboside mimics the effects of insulin on the expression of the 2 key gluconeogenic genes PEPCK and glucose-6-phosphatase

7 January 2021

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Inhibitory effect of a marine-sponge toxin, okadaic acid, on protein phosphatases. Specificity and kinetics

7 January 2021

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Inactivation of glycogen synthase kinase-3 beta by phosphorylation: new kinase connections in insulin and growth-factor signalling

7 January 2021

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10.1016/S0014-5793(03)00774-9

7 January 2021

Identifiers

DOI

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4 December 2017

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