Ectopic expression of nonliganded retinoic acid receptor beta abrogates AP-1 activity by selective degradation of c-Jun in cervical carcinoma cells (Q45016647)

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English	Ectopic expression of nonliganded retinoic acid receptor beta abrogates AP-1 activity by selective degradation of c-Jun in cervical carcinoma cells	scientific article

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scholarly article

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Europe PubMed Central

8 December 2017

Ectopic expression of nonliganded retinoic acid receptor beta abrogates AP-1 activity by selective degradation of c-Jun in cervical carcinoma cells (English)

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Europe PubMed Central

8 December 2017

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Harald zur Hausen

5

object named as

Harald zur Hausen

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Europe PubMed Central

8 December 2017

2

object named as

Ubaldo Soto

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Europe PubMed Central

8 December 2017

Jan van Riggelen

3

object named as

Jan van Riggelen

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Europe PubMed Central

8 December 2017

author name string

Johanna De-Castro Arce

1

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Europe PubMed Central

8 December 2017

Elisabeth Schwarz

4

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Europe PubMed Central

8 December 2017

Frank Rösl

6

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Europe PubMed Central

8 December 2017

language of work or name

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publication date

11 August 2004

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Europe PubMed Central

8 December 2017

Journal of Biological Chemistry

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Europe PubMed Central

8 December 2017

279

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Europe PubMed Central

8 December 2017

44

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Europe PubMed Central

8 December 2017

45408-45416

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Europe PubMed Central

8 December 2017

The promise of retinoids to fight against cancer.

1 reference

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Retinoid X receptor is an auxiliary protein for thyroid hormone and retinoic acid receptors

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Homodimer formation of retinoid X receptor induced by 9-cis retinoic acid

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Synergistic activation of retinoic acid (RA)-responsive genes and induction of embryonal carcinoma cell differentiation by an RA receptor alpha (RAR alpha)-, RAR beta-, or RAR gamma-selective ligand in combination with a retinoid X receptor-specific

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Coactivator and corepressor complexes in nuclear receptor function

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Retinoid receptors in health and disease: co-regulators and the chromatin connection

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Active repression by unliganded retinoid receptors in development: less is sometimes more

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Cellular control of human papillomavirus oncogene transcription

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21 January 2018

AP-1: one switch for many signals

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21 January 2018

AP-1 transrepressing retinoic acid does not deplete coactivators or AP-1 monomers but may target specific Jun or Fos containing dimers

1 reference

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21 January 2018

Ligand-activated retinoic acid receptor inhibits AP-1 transactivation by disrupting c-Jun/c-Fos dimerization.

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21 January 2018

A CBP integrator complex mediates transcriptional activation and AP-1 inhibition by nuclear receptors

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21 January 2018

Loss of retinoic acid receptor-beta expression is an early event during esophageal carcinogenesis

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Methylation and silencing of the retinoic acid receptor-beta 2 gene in cervical cancer

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RAR beta 2-mediated growth inhibition in HeLa cells

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Tumor-suppressive effect of the retinoic acid receptor beta in human epidermoid lung cancer cells

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Conversion of HPV 18 positive non-tumorigenic HeLa-fibroblast hybrids to invasive growth involves loss of TNF-alpha mediated repression of viral transcription and modification of the AP-1 transcription complex.

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Differential activation of ERK and JNK mitogen-activated protein kinases by Raf-1 and MEKK

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Identification of a dual specificity kinase that activates the Jun kinases and p38-Mpk2

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Resistance to diverse drugs and ultraviolet light conferred by overexpression of a novel human 26 S proteasome subunit

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Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells

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Transient induction of metallothionein isoform 3 (MT-3), c-fos, c-jun and c-myc in human proximal tubule cells exposed to cadmium

1 reference

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21 January 2018

All-trans, 13-cis and 9-cis retinoic acids induce a fully reversible growth inhibition in HNSCC cell lines: implications for in vivo retinoic acid use.

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21 January 2018

Blocking activator protein-1 activity, but not activating retinoic acid response element, is required for the antitumor promotion effect of retinoic acid

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21 January 2018

Retinoic acid is a negative regulator of AP-1-responsive genes.

1 reference

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21 January 2018

Degradation of c-Fos by the 26S proteasome is accelerated by c-Jun and multiple protein kinases

1 reference

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21 January 2018

Differential expression of retinoic acid receptor beta (RARbeta) and the AP-1 transcription factor in normal, premalignant and malignant human laryngeal tissues

1 reference

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21 January 2018

c-Jun NH2-terminal kinases target the ubiquitination of their associated transcription factors

1 reference

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21 January 2018

Tumor necrosis factor alpha-induced activation of c-jun N-terminal kinase is mediated by TRAF2.

1 reference

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AP-1 function and regulation

1 reference

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Ubiquitination and degradation of ATF2 are dimerization dependent

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Unexpected transcriptional induction of monocyte chemoattractant protein 1 by proteasome inhibition: involvement of the c-Jun N-terminal kinase-activator protein 1 pathway

1 reference

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21 January 2018

The c-Jun N-terminal protein kinase family of mitogen-activated protein kinases (JNK MAPKs).

1 reference

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Reduced ubiquitin-dependent degradation of c-Jun after phosphorylation by MAP kinases

1 reference

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Degradation of retinoid X receptor alpha by TPA through proteasome pathway in gastric cancer cells

1 reference

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The AF-1 and AF-2 domains of RAR gamma 2 and RXR alpha cooperate for triggering the transactivation and the degradation of RAR gamma 2/RXR alpha heterodimers

1 reference

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21 January 2018

The regulation of AP-1 activity by mitogen-activated protein kinases

1 reference

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Mixed-lineage kinase control of JNK and p38 MAPK pathways

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Disturbance of tumor necrosis factor alpha-mediated beta interferon signaling in cervical carcinoma cells

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All-trans-retinoic acid inhibits Jun N-terminal kinase by increasing dual-specificity phosphatase activity

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Transcriptional induction of mitogen-activated protein kinase phosphatase 1 by retinoids. Selective roles of nuclear receptors and contribution to the antiapoptotic effect

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MEK kinase 1 (MEKK1) transduces c-Jun NH2-terminal kinase activation in response to changes in the microtubule cytoskeleton.

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Independent human MAP-kinase signal transduction pathways defined by MEK and MKK isoforms

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The 26S proteasome: a molecular machine designed for controlled proteolysis

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Are there multiple proteolytic pathways contributing to c-Fos, c-Jun and p53 protein degradation in vivo?

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21 January 2018

Ubiquitin-dependent c-Jun degradation in vivo is mediated by the delta domain

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Cellular aging and transformation suppression: a role for retinoic acid receptor beta 2.

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Promoter Methylation and Silencing of the Retinoic Acid Receptor- Gene in Lung Carcinomas

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A human lymphoid- specific transcription factor that activates immunoglobulin genes is a homoeobox protein

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Phosphoproteome dynamics reveal novel ERK1/2 MAP kinase substrates with broad spectrum of functions

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Dual specificity phosphatases: a gene family for control of MAP kinase function.

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All-trans-retinoic acid inhibits Jun N-terminal kinase-dependent signaling pathways

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Stress-activated kinases regulate protein stability

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Purified transcription factor AP-1 interacts with TPA-inducible enhancer elements

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Regulation and composition of activator protein 1 (AP-1) transcription factors controlling collagenase and c-Jun promoter activities

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10.1074/JBC.M401818200

1 reference

Europe PubMed Central

8 December 2017

1 reference

Europe PubMed Central

8 December 2017

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