Oncogenic activation of c-Myb correlates with a loss of negative regulation by TIF1beta and Ski. (Q45130667)

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18 November 2019

title

Oncogenic activation of c-Myb correlates with a loss of negative regulation by TIF1beta and Ski (English)

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18 November 2019

8

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18 November 2019

author name string

Teruaki Nomura

1

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18 November 2019

Jun Tanikawa

2

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18 November 2019

Hiroshi Akimaru

3

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18 November 2019

Chie Kanei-Ishii

4

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18 November 2019

Emi Ichikawa-Iwata

5

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18 November 2019

Md Matiullah Khan

6

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18 November 2019

Hiroki Ito

7

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18 November 2019

language of work or name

English

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publication date

3 February 2004

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Journal of Biological Chemistry

18 November 2019

published in

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18 November 2019

volume

279

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18 November 2019

issue

16

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18 November 2019

page(s)

16715-16726

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Nucleotide sequence of the retroviral leukemia gene v-myb and its cellular progenitor c-myb: The architecture of a transduced oncogene

18 November 2019

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A functional c-myb gene is required for normal murine fetal hepatic hematopoiesis

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c-Myb is essential for early T cell development

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Drosophila myb is required for the G2/M transition and maintenance of diploidy

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Myb controls G(2)/M progression by inducing cyclin B expression in the Drosophila eye imaginal disc

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The homeobox gene GBX2, a target of the myb oncogene, mediates autocrine growth and monocyte differentiation

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v-Myb of E26 leukemia virus up-regulates bcl-2 and suppresses apoptosis in myeloid cells

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A dominant interfering Myb mutant causes apoptosis in T cells

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Solution structure of a specific DNA complex of the Myb DNA-binding domain with cooperative recognition helices

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Ski is involved in transcriptional regulation by the repressor and full-length forms of Gli3

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Carboxy-terminal elements of c-Myb negatively regulate transcriptional activation in cis and in trans

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Transactivation and transformation by Myb are negatively regulated by a leucine-zipper structure

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Ski is a component of the histone deacetylase complex required for transcriptional repression by Mad and thyroid hormone receptor

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A complex containing N-CoR, mSin3 and histone deacetylase mediates transcriptional repression

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Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression

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Nuclear receptor repression mediated by a complex containing SMRT, mSin3A, and histone deacetylase

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Histone deacetylases and SAP18, a novel polypeptide, are components of a human Sin3 complex

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Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor

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Interaction of the Ski oncoprotein with Smad3 regulates TGF-beta signaling

21 January 2018

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The Ski oncoprotein interacts with the Smad proteins to repress TGFbeta signaling

21 January 2018

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Increased susceptibility to tumorigenesis of ski-deficient heterozygous mice

21 January 2018

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Identification of unknown target genes of human transcription factors using chromatin immunoprecipitation

21 January 2018

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Bonus, a Drosophila homolog of TIF1 proteins, interacts with nuclear receptors and can inhibit betaFTZ-F1-dependent transcription

21 January 2018

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KAP-1, a novel corepressor for the highly conserved KRAB repression domain

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Interaction with members of the heterochromatin protein 1 (HP1) family and histone deacetylation are differentially involved in transcriptional silencing by members of the TIF1 family

21 January 2018

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21 January 2018

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Comparison of the free and DNA-complexed forms of the DNA-binding domain from c-Myb

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21 January 2018

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Molecular cloning reveals that the p160 Myb-binding protein is a novel, predominantly nucleolar protein which may play a role in transactivation by Myb

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.M313069200

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14761981%20AND%20SRC:MED&resulttype=core&format=json

18 November 2019

1 reference