Retinoic acid stabilizes p27Kip1 in EBV-immortalized lymphoblastoid B cell lines through enhanced proteasome-dependent degradation of the p45Skp2 and Cks1 proteins. (Q45286150)

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scientific article published in April 2005

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English	Retinoic acid stabilizes p27Kip1 in EBV-immortalized lymphoblastoid B cell lines through enhanced proteasome-dependent degradation of the p45Skp2 and Cks1 proteins.	scientific article published in April 2005

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8 January 2020

Retinoic acid stabilizes p27Kip1 in EBV-immortalized lymphoblastoid B cell lines through enhanced proteasome-dependent degradation of the p45Skp2 and Cks1 proteins (English)

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8 January 2020

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Riccardo Dolcetti

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8 January 2020

Jessica Dal Col

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8 January 2020

Massimo Guidoboni

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8 January 2020

Roberta Maestro

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8 January 2020

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Paola Zancai

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Mauro Boiocchi

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8 January 2020

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Silvana Rizzo

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8 January 2020

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Sara Piccinin

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8 January 2020

Roberta Cariati

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1 April 2005

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8 January 2020

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8 January 2020

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8 January 2020

2483-2494

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8 January 2020

Deregulated degradation of the cdk inhibitor p27 and malignant transformation.

1 reference

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Cyclin D1 proteolysis: a retinoid chemoprevention signal in normal, immortalized, and transformed human bronchial epithelial cells

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Retinoic acid-mediated growth arrest requires ubiquitylation and degradation of the F-box protein Skp2.

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Leptomycin B inhibition of signal-mediated nuclear export by direct binding to CRM1

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Phosphorylation of p27Kip1 on serine 10 is required for its binding to CRM1 and nuclear export

1 reference

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Phosphorylation at serine 10, a major phosphorylation site of p27(Kip1), increases its protein stability

1 reference

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21 January 2018

Posttranslational regulation of cyclin D1 by retinoic acid: a chemoprevention mechanism

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Role of the F-box protein Skp2 in lymphomagenesis

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Transcriptional activation of the human p21(WAF1/CIP1) gene by retinoic acid receptor. Correlation with retinoid induction of U937 cell differentiation

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Retinoic acid decreases targeting of p27 for degradation via an N-myc-dependent decrease in p27 phosphorylation and an N-myc-independent decrease in Skp2.

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21 January 2018

Retinoic acid promotes ubiquitination and proteolysis of cyclin D1 during induced tumor cell differentiation

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The cytoplasmic shuttling and subsequent degradation of p27Kip1 mediated by Jab1/CSN5 and the COP9 signalosome complex

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Retinoic acid-induced cell cycle arrest of human myeloid cell lines is associated with sequential down-regulation of c-Myc and cyclin E and posttranscriptional up-regulation of p27(Kip1).

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MaRX: an approach to genetics in mammalian cells.

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Cks1 is degraded via the ubiquitin‐proteasome pathway in a cell cycle‐dependent manner

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Retinoic acid-mediated G1 arrest is associated with induction of p27(Kip1) and inhibition of cyclin-dependent kinase 3 in human lung squamous carcinoma CH27 cells

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Expression of Skp2, a p27(Kip1) ubiquitin ligase, in malignant lymphoma: correlation with p27(Kip1) and proliferation index

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Cdk2-dependent phosphorylation of p27 facilitates its Myc-induced release from cyclin E/cdk2 complexes

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Degradation of the cyclin-dependent-kinase inhibitor p27Kip1 is instigated by Jab1

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Retinoic acid-mediated growth arrest of EBV-immortalized B lymphocytes is associated with multiple changes in G1 regulatory proteins: p27Kip1 up-regulation is a relevant early event

1 reference

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Cell cycle genes as targets of retinoid induced ovarian tumor cell growth suppression

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p19Skp1 and p45Skp2 are essential elements of the cyclin A-CDK2 S phase kinase

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10.1038/SJ.ONC.1208458

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8 January 2020

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8 January 2020

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