Sodium channel beta4 subunit: down-regulation and possible involvement in neuritic degeneration in Huntington's disease transgenic mice (Q45301710)

11 December 2017

title

Sodium channel beta4 subunit: down-regulation and possible involvement in neuritic degeneration in Huntington's disease transgenic mice (English)

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11 December 2017

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11 December 2017

Haruko Miyazaki

2

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Naoaki Sakamoto

3

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Celine Becquet

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Yoko Machida

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Kumi Kaneko

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Chiharu Uchikawa

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Taishi Suzuki

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Masaru Kurosawa

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Tetsurou Ikeda

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Akira Tamaoka

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Takashi Sakurai

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Nobuyuki Nukina

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11 December 2017

language of work or name

English

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publication date

1 July 2006

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Journal of Neurochemistry

11 December 2017

published in

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volume

98

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issue

2

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page(s)

518-529

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From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

11 December 2017

cites work

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Reduced sodium channel density, altered voltage dependence of inactivation, and increased susceptibility to seizures in mice lacking sodium channel beta 2-subunits

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Mice lacking sodium channel beta1 subunits display defects in neuronal excitability, sodium channel expression, and nodal architecture

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Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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Sodium channel beta1 subunits promote neurite outgrowth in cerebellar granule neurons

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Early degenerative changes in transgenic mice expressing mutant huntingtin involve dendritic abnormalities but no impairment of mitochondrial energy production

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

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Structure and function of the beta 2 subunit of brain sodium channels, a transmembrane glycoprotein with a CAM motif

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Presenilin/gamma-secretase-mediated cleavage of the voltage-gated sodium channel beta2-subunit regulates cell adhesion and migration

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Decreased expression of striatal signaling genes in a mouse model of Huntington's disease

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Sodium channel beta subunits mediate homophilic cell adhesion and recruit ankyrin to points of cell-cell contact

21 January 2018

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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

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beta 3: an additional auxiliary subunit of the voltage-sensitive sodium channel that modulates channel gating with distinct kinetics

21 January 2018

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The expression of the mouse Zic1, Zic2, and Zic3 gene suggests an essential role for Zic genes in body pattern formation

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Differential modulation of sodium channel gating and persistent sodium currents by the beta1, beta2, and beta3 subunits.

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A sodium channel signaling complex: modulation by associated receptor protein tyrosine phosphatase beta

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Dendritic spine pathology and deficits in experience-dependent dendritic plasticity in R6/1 Huntington's disease transgenic mice

21 January 2018

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Interaction of voltage-gated sodium channels with the extracellular matrix molecules tenascin-C and tenascin-R

21 January 2018

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Transcriptional abnormalities in Huntington disease

21 January 2018

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beta Subunits of voltage-gated sodium channels are novel substrates of beta-site amyloid precursor protein-cleaving enzyme (BACE1) and gamma-secretase

21 January 2018

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21 January 2018

Identifiers

DOI

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11 December 2017

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