Soluble TNF-α receptor secretion from healthy or dystrophic mice after AAV6-mediated muscle gene transfer (Q45861904)

17 December 2017

title

Soluble TNF-α receptor secretion from healthy or dystrophic mice after AAV6-mediated muscle gene transfer (English)

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17 December 2017

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G Moulay

1

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17 December 2017

C Masurier

2

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17 December 2017

P Bigey

3

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17 December 2017

D Scherman

4

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17 December 2017

A Kichler

5

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17 December 2017

publication date

1 July 2010

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17 December 2017

published in

Gene Therapy

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17 December 2017

volume

17

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17 December 2017

page(s)

1400-1410

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17 December 2017

issue

11

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17 December 2017

https://scigraph.springernature.com/pub.10.1038/gt.2010.94

exact match

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cites work

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TNF-alpha acts via p38 MAPK to stimulate expression of the ubiquitin ligase atrogin1/MAFbx in skeletal muscle

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Anti-TNFalpha (Remicade) therapy protects dystrophic skeletal muscle from necrosis.

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Viral serotype and the transgene sequence influence overlapping adeno-associated viral (AAV) vector-mediated gene transfer in skeletal muscle

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Stable gene transfer and expression of human blood coagulation factor IX after intramuscular injection of recombinant adeno-associated virus

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Posttranslational modifications of recombinant myotube-synthesized human factor IX.

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Sustained secretion of human alpha-1-antitrypsin from murine muscle transduced with adeno-associated virus vectors

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Antiangiogenic effect of interleukin-10 in ischemia-induced angiogenesis in mice hindlimb

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Gene therapy of collagen-induced arthritis by electrotransfer of human tumor necrosis factor-alpha soluble receptor I variants

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Role of tumour necrosis factor alpha, but not of cyclo-oxygenase-2-derived eicosanoids, on functional and morphological indices of dystrophic progression in mdx mice: a pharmacological approach.

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Immune evasion by muscle-specific gene expression in dystrophic muscle

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Systemic protein delivery by muscle-gene transfer is limited by a local immune response

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Anti-monocyte chemoattractant protein-1 gene therapy inhibits vascular remodeling in rats: blockade of MCP-1 activity after intramuscular transfer of a mutant gene inhibits vascular remodeling induced by chronic blockade of NO synthesis

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Adeno-associated virus vector-mediated gene transfer into dystrophin-deficient skeletal muscles evokes enhanced immune response against the transgene product

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Interleukin-10 expression after intramuscular DNA electrotransfer: kinetic studies

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Plasmid electrotransfer of eye ciliary muscle: principles and therapeutic efficacy using hTNF-alpha soluble receptor in uveitis

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Common pathological mechanisms in mouse models for muscular dystrophies

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Expression of tumor necrosis factor-alpha in regenerating muscle fibers in inflammatory and non-inflammatory myopathies.

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Reduced necrosis of dystrophic muscle by depletion of host neutrophils, or blocking TNFalpha function with Etanercept in mdx mice

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Sustained AAV-mediated dystrophin expression in a canine model of Duchenne muscular dystrophy with a brief course of immunosuppression

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7 January 2021

Identifiers

DOI

10.1038/GT.2010.94

1 reference

17 December 2017

Dimensions Publication ID

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1 reference

17 December 2017