Prognostic significance of c-Met expression in glioblastomas (Q46170165)

20 December 2017

title

Prognostic significance of c-Met expression in glioblastomas (English)

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20 December 2017

8

Yeon-Lim Suh

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20 December 2017

author name string

Doo-Sik Kong

1

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20 December 2017

Sang-Yong Song

2

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20 December 2017

Duk-Hwan Kim

3

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20 December 2017

Kyeung Min Joo

4

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20 December 2017

Jin-San Yoo

5

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20 December 2017

Jong Sung Koh

6

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20 December 2017

Seung Myung Dong

7

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Jung-Il Lee

9

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20 December 2017

Kwan Park

10

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20 December 2017

Jong Hyun Kim

11

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20 December 2017

Do-Hyun Nam

12

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20 December 2017

language of work or name

English

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publication date

1 January 2009

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20 December 2017

number of pages

9

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inferred from page(s)

published in

Cancer

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20 December 2017

volume

115

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20 December 2017

issue

1

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20 December 2017

page(s)

140-148

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20 December 2017

Met, metastasis, motility and more

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Scatter factor/hepatocyte growth factor stimulation of glioblastoma cell cycle progression through G(1) is c-Myc dependent and independent of p27 suppression, Cdk2 activation, or E2F1-dependent transcription

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21 January 2018

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Scatter factor protects epithelial and carcinoma cells against apoptosis induced by DNA-damaging agents

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Targeting the c-Met pathway potentiates glioblastoma responses to gamma-radiation

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Clinical and radiographic response in a minority of patients with recurrent malignant gliomas treated with high-dose tamoxifen

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Suppression of Ras-mediated tumorigenicity and metastasis through inhibition of the Met receptor tyrosine kinase

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Activated ras and ret oncogenes induce over-expression of c-met (hepatocyte growth factor receptor) in human thyroid epithelial cells

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Hepatocyte growth factor-induced expression of ornithine decarboxylase, c-met, and c-myc is differently affected by protein kinase inhibitors in human hepatoma cells HepG2.

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MT1-MMP initiates activation of pro-MMP-2 and integrin alphavbeta3 promotes maturation of MMP-2 in breast carcinoma cells

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7 January 2021

In vivo targeting of SF/HGF and c-met expression via U1snRNA/ribozymes inhibits glioma growth and angiogenesis and promotes apoptosis

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The cytokine hepatocyte growth factor/scatter factor inhibits apoptosis and enhances DNA repair by a common mechanism involving signaling through phosphatidyl inositol 3' kinase

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Multifocal glioblastoma multiforme: prognostic factors and patterns of progression

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7 January 2021

Prognostic significance of intracranial dissemination of glioblastoma multiforme in adults

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7 January 2021

Hepatocyte growth factor-stimulated renal tubular mitogenesis: effects on expression of c-myc, c-fos, c-met, VEGF and the VHL tumour-suppressor and related genes

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7 January 2021

Expression and tissue localization of membrane-type 1, 2, and 3 matrix metalloproteinases in human astrocytic tumors

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7 January 2021

Identifiers

DOI

10.1002/CNCR.23972

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20 December 2017

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20 December 2017