Pathophysiological consequences of VEGF-induced vascular permeability (Q46349878)

21 December 2017

title

Pathophysiological consequences of VEGF-induced vascular permeability (English)

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21 December 2017

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Sara M Weis

1

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21 December 2017

David A Cheresh

series ordinal

2

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21 December 2017

language of work or name

English

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publication date

1 September 2005

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21 December 2017

published in

Nature

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21 December 2017

volume

437

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21 December 2017

page(s)

497-504

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21 December 2017

issue

7058

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21 December 2017

https://scigraph.springernature.com/pub.10.1038/nature03987

exact match

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VE-PTP and VE-cadherin ectodomains interact to facilitate regulation of phosphorylation and cell contacts

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Contact inhibition of VEGF-induced proliferation requires vascular endothelial cadherin, beta-catenin, and the phosphatase DEP-1/CD148

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SHP2 association with VE-cadherin complexes in human endothelial cells is regulated by thrombin

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The vascular endothelial growth factor receptor KDR activates multiple signal transduction pathways in porcine aortic endothelial cells

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VEGF gene delivery to myocardium: deleterious effects of unregulated expression

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Direct evidence that the VEGF-specific antibody bevacizumab has antivascular effects in human rectal cancer

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Circulating angiogenesis regulators in cancer patients.

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Nicotine and cotinine up-regulate vascular endothelial growth factor expression in endothelial cells

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Second hand smoke stimulates tumor angiogenesis and growth

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Identifiers

DOI

10.1038/NATURE03987

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21 December 2017

PubMed ID

16177780

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21 December 2017