Rosiglitazone and 15-deoxy-Delta12,14-prostaglandin J2 cause potent neuroprotection after experimental stroke through noncompletely overlapping mechanisms. (Q46611073)

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scientific article published in February 2006

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English	Rosiglitazone and 15-deoxy-Delta12,14-prostaglandin J2 cause potent neuroprotection after experimental stroke through noncompletely overlapping mechanisms.	scientific article published in February 2006

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31 December 2019

Rosiglitazone and 15-deoxy-Delta12,14-prostaglandin J2 cause potent neuroprotection after experimental stroke through noncompletely overlapping mechanisms (English)

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31 December 2019

(RS)-rosiglitazone

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31 December 2019

Ignacio Lizasoain

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31 December 2019

María Ángeles Moro Sánchez

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31 December 2019

Marta P Pereira

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31 December 2019

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31 December 2019

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31 December 2019

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Antonio Cárdenas

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31 December 2019

Antoni Dávalos

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31 December 2019

José Vivancos

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31 December 2019

Joaquín Serena

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31 December 2019

Pedro Lorenzo

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31 December 2019

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1 February 2006

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31 December 2019

Journal of Cerebral Blood Flow & Metabolism

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31 December 2019

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31 December 2019

2

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31 December 2019

218-229

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31 December 2019

The mechanisms of action of PPARs

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15-deoxy-delta12,14-prostaglandin J2 regulates the functional state and the survival of microglial cells through multiple molecular mechanisms.

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Unriddling the role of matrix metalloproteinases in human cerebral stroke

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Inhibition of IkappaB kinase and IkappaB phosphorylation by 15-deoxy-Delta(12,14)-prostaglandin J(2) in activated murine macrophages

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15d-PGJ2 and rosiglitazone suppress Janus kinase-STAT inflammatory signaling through induction of suppressor of cytokine signaling 1 (SOCS1) and SOCS3 in glia

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Molecular basis for the direct inhibition of AP-1 DNA binding by 15-deoxy-Delta 12,14-prostaglandin J2.

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