The prognostic significance of Jun transcription factors in ovarian cancer (Q47784628)

5 February 2018

title

The prognostic significance of Jun transcription factors in ovarian cancer (English)

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6

Karin Milde-Langosch

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author name string

Kerstin Eckhoff

1

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Roland Flurschütz

2

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Fabian Trillsch

3

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Sven Mahner

4

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Fritz Jänicke

5

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publication date

13 August 2013

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Journal of Cancer Research and Clinical Oncology

published in

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5 February 2018

volume

139

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issue

10

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page(s)

1673-1680

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5 February 2018

Transcription factor JunD, deprived of menin, switches from growth suppressor to growth promoter

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Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

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Close encounters of many kinds: Fos-Jun interactions that mediate transcription regulatory specificity.

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Signal transduction by the JNK group of MAP kinases

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JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

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Cancer incidence and mortality patterns in Europe: estimates for 40 countries in 2012

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JunD reduces tumor angiogenesis by protecting cells from oxidative stress

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AP-1 in mouse development and tumorigenesis

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The regulation of AP-1 activity by mitogen-activated protein kinases

21 January 2018

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Phosphorylated c-Jun and Fra-1 induce matrix metalloproteinase-1 and thereby regulate invasion activity of 143B osteosarcoma cells

21 January 2018

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Withdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell death

21 January 2018

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C-Fos expression is a molecular predictor of progression and survival in epithelial ovarian carcinoma

21 January 2018

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Aberrantly expressed c-Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF-kappa B.

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The Fos family of transcription factors and their role in tumourigenesis

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Role of Fra-2 in breast cancer: influence on tumor cell invasion and motility

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Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras.

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AP-1--The Jun proteins: Oncogenes or tumor suppressors in disguise?

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Reactivation of proliferin gene expression is associated with increased angiogenesis in a cell culture model of fibrosarcoma tumor progression

21 January 2018

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c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer

21 January 2018

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JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun

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JunD protects cells from p53-dependent senescence and apoptosis.

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c-Jun regulates cell cycle progression and apoptosis by distinct mechanisms

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High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model.

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Role of c-Fos/JunD in protecting stress-induced cell death

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12 December 2020

2004 consensus statements on the management of ovarian cancer: final document of the 3rd International Gynecologic Cancer Intergroup Ovarian Cancer Consensus Conference (GCIG OCCC 2004).

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A null mutation at the c-jun locus causes embryonic lethality and retarded cell growth in culture

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12 December 2020

The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation

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12 December 2020

Expression of Jun and Fos proteins in ovarian tumors of different malignant potential and in ovarian cancer cell lines.

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12 December 2020

10.1007/S00432-013-1489-Y

Identifiers

DOI

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5 February 2018

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5 February 2018